Consequences of Gastric Disease - Pathology - Revision history

Bara: Text replace - "[[Forestomach - Anatomy & Physiology|" to "[[Monogastric Stomach - Anatomy & Physiology|"

2010-09-06T13:15:37Z

Text replace - "[[Forestomach - Anatomy & Physiology|" to "[[Monogastric Stomach - Anatomy & Physiology|"

Bara at 12:09, 26 July 2010

2010-07-26T12:09:51Z

Bara at 22:58, 28 June 2010

2010-06-28T22:58:08Z

Bara at 14:19, 29 May 2010

2010-05-29T14:19:26Z

Bara: moved Stomach and Abomasum Consequences of Gastric Disease - Pathology to Consequences of Gastric Disease - Pathology

2010-05-29T14:19:03Z

moved Stomach and Abomasum Consequences of Gastric Disease - Pathology to Consequences of Gastric Disease - Pathology

Jtulloch at 14:00, 26 September 2008

2008-09-26T14:00:33Z

A.allison at 23:28, 25 August 2008

2008-08-25T23:28:16Z

A.allison at 23:03, 25 August 2008

2008-08-25T23:03:29Z

A.allison: New page: {{toplink |backcolour =BCED91 |linkpage =Alimentary System - Pathology |linktext =Alimentary System |maplink = Alimentary System (Content Map) - Pathology |pagetype =Pathology |sublink1=St...

2008-08-25T23:01:31Z

New page: {{toplink |backcolour =BCED91 |linkpage =Alimentary System - Pathology |linktext =Alimentary System |maplink = Alimentary System (Content Map) - Pathology |pagetype =Pathology |sublink1=St...

New page

{{toplink
|backcolour =BCED91
|linkpage =Alimentary System - Pathology
|linktext =Alimentary System
|maplink = Alimentary System (Content Map) - Pathology
|pagetype =Pathology
|sublink1=Stomach and Abomasum - Pathology
|subtext1=STOMACH AND ABOMASUM
}}
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 * Causes failure of digestion.
 ** Anorexia and weight loss follow.
-* Increases the number of bacteria in the [[Forestomach - Anatomy & Physiology|stomach]].
+* Increases the number of bacteria in the [[Monogastric Stomach - Anatomy & Physiology|stomach]].
 * Diarrhoea reults
 ** Cause is unknwn is unknown but appears to be related to the elevated pH.

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 {{review}}
-==Vomiting==
+==[[Vomiting]]==
 ==Raised Intraluminal pH==

← Older revision		Revision as of 22:58, 28 June 2010
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	** Due to bleeding from gastric ulceration.		** Due to bleeding from gastric ulceration.
	[[Category:Stomach_and_Abomasum_-_Pathology]]		[[Category:Stomach_and_Abomasum_-_Pathology]]
		+	[[Category:To_Do_-_Clinical]]

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 {{review}}
 ==Vomiting==
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 * Is usually haemorrhagic in nature
 ** Due to bleeding from gastric ulceration.

← Older revision		Revision as of 14:00, 26 September 2008
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		+	{{review}}
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	\|backcolour =BCED91		\|backcolour =BCED91

← Older revision	Revision as of 14:19, 29 May 2010
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 *** [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex|Vomiting]] also induces hypochloraemia, meaning bicarbonate rather than chloride is resorbed with the Na+ to maintain electrical neutrality
 **** This perpetuates the alkalosis.
-* [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex|Vomiting]] does not occur in the ruminant although [[The Abomasum|abomasal]] content may reflux into the [[Stomach and Abomasum - Anatomy & Physiology|forestomachs]].
+* [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex|Vomiting]] does not occur in the ruminant although [[The Abomasum - Anatomy & Physiology|abomasal]] content may reflux into the [[Stomach and Abomasum - Anatomy & Physiology|forestomachs]].
-** Sequestration of secretions in the [[The Abomasum|abomasum]] will have similar effects to pyloric outflow obstruction with [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex|vomiting]] in the monogastric animal.
+** Sequestration of secretions in the [[The Abomasum - Anatomy & Physiology|abomasum]] will have similar effects to pyloric outflow obstruction with [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex|vomiting]] in the monogastric animal.
 *** e.g. abomasal torsion
 *** Causes dehydration, hypochloraemia, hypokalaemia and metabolic alkalosis.

← Older revision		Revision as of 23:03, 25 August 2008
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	<br>		<br>
		+	==Vomiting==
		+
		+	* Has potentially lethal effects in the monogastric animal.
		+
		+
		+	===Water Loss===
		+
		+	* Fluid loss is evident as:
		+	*# An increased PCV or haematocrit.
		+	*# An increased total protein concentration.
		+	*# A prerenal azotaemia.
		+
		+
		+	===Gastric Electrolyte Loss===
		+
		+	* The main losses are of H<sup>+</sup> and Cl<sup>-</sup>, and also K<sup>+</sup>
		+	* Can potentially cause metabolic alkalosis, although this is only likely with disease which stops at the pylorus, e.g.: pyloric outflow obstruction.
		+	** In cases where mild alkalosis occurs, homeostatic mechanisms produce a more alkaline urine to restore normal body pH.
		+	** However, in severe metablolic alkalosis with marked dehydration, acidic urine may be produced- this is termed '''paradoxical aciduria'''.
		+	*** Because [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex\|vomiting]] induceses hypokalaemia, there is an overriding stimulus in the kidney for Na<sup>+</sup> (and therefore water) retention.
		+	*** Na+ can only be resorbed in exchange for H+
		+	**** H<sup>+</sup> is therefore excreted in the urine, causing it to be acidic.
		+	*** [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex\|Vomiting]] also induces hypochloraemia, meaning bicarbonate rather than chloride is resorbed with the Na+ to maintain electrical neutrality
		+	**** This perpetuates the alkalosis.
		+	* [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex\|Vomiting]] does not occur in the ruminant although [[The Abomasum\|abomasal]] content may reflux into the [[Stomach and Abomasum - Anatomy & Physiology\|forestomachs]].
		+	** Sequestration of secretions in the [[The Abomasum\|abomasum]] will have similar effects to pyloric outflow obstruction with [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex\|vomiting]] in the monogastric animal.
		+	*** e.g. abomasal torsion
		+	*** Causes dehydration, hypochloraemia, hypokalaemia and metabolic alkalosis.
		+
		+
		+	* Lesions in the [[Small Intestine - Anatomy & Physiology\|small intestine]] can also lead to [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex\|vomiting]]
		+	** Both gastric acid and pancreatic and intestinal bicarbonate secretions are lost
		+	*** Animal consequently has a normal pH or may even be acidotic.
		+
		+	==Raised Intraluminal pH==
		+
		+	* Associated with some forms of gastritis.
		+	** e.g. Ostertagiasis
		+	* Causes failure of digestion.
		+	** Anorexia and weight loss follow.
		+	* Increases the number of bacteria in the [[Forestomach - Anatomy & Physiology\|stomach]].
		+	* Diarrhoea reults
		+	** Cause is unknwn is unknown but appears to be related to the elevated pH.
		+
		+	== Hyperacidity==
		+
		+	* May develop in certain gastric disturbances
		+	* Thought to be a contributory factor in peptic ulceration.
		+
		+	==Anaemia==
		+
		+	* May also develop in certain gastric diseases
		+	* Is usually haemorrhagic in nature
		+	** Due to bleeding from gastric ulceration.