Endocrinology Quiz - Revision history

Ctrace at 11:01, 26 June 2011

2011-06-26T11:01:39Z

Bara: Text replace - "-_Anatomy_%26_Physiology" to "- Anatomy & Physiology"

2010-11-29T15:21:13Z

Text replace - "-_Anatomy_%26_Physiology" to "- Anatomy & Physiology"

Suzannah.stacey: Text replace - "Calcium Homeostasis - Anatomy & Physiology" to "Calcium"

2010-11-05T13:57:16Z

Text replace - "Calcium Homeostasis - Anatomy & Physiology" to "Calcium"

Bara at 14:29, 13 September 2010

2010-09-13T14:29:51Z

Bara: Text replace - "[[Small Intestine - Anatomy & Physiology#" to "[[Small Intestine Overview - Anatomy & Physiology#"

2010-09-07T12:59:30Z

Text replace - "[[Small Intestine - Anatomy & Physiology#" to "[[Small Intestine Overview - Anatomy & Physiology#"

Bevpanto at 18:18, 26 January 2010

2010-01-26T18:18:16Z

Bevpanto at 18:16, 26 January 2010

2010-01-26T18:16:34Z

Bevpanto at 18:15, 26 January 2010

2010-01-26T18:15:38Z

Bevpanto at 17:46, 26 January 2010

2010-01-26T17:46:32Z

Bevpanto at 17:46, 26 January 2010

2010-01-26T17:46:00Z

@@ Line 3: / Line 3: @@
 |linktext = WikiQuiz
 |pagetype=Quiz
-|Review=''' David Gardner''' BSc (Hons) PhD Associate Professor in developmental physiology <br>   '''Alison Mostyn''' BSc (Hons) PhD Lecturer in Comparative Cellular Physiology}}{{QuizCat|topic=Anatomy|topicsubcategory=E|system=Endocrine|systemsubcategory=E}}<br>
+|Review=''' David Gardner''' BSc (Hons) PhD Associate Professor in developmental physiology <br>   '''Alison Mostyn''' BSc (Hons) PhD Lecturer in Comparative Cellular Physiology}}<br>
@@ Line 231: / Line 231: @@
 image= "">
 </WikiQuiz>

@@ Line 79: / Line 79: @@
 choice2="Thyroid Stimulating Hormone (TSH) / Somatostatin"
 correctchoice="5"
-feedback5="'''Correct!''' Thyrotrophin releasing hormone (TRH) from the hypothalamus stimulates the release of thyroid stimulating hormone (TSH) from the anterior pituitary which in turn stimulates the release of thyroid hormone form the thyroid gland. Somatostatin inhibits the release of TSH. [[Thyroid_Gland_-_Anatomy_%26_Physiology#Regulationn|WikiVet Article: Thyroid physiology]]"
+feedback5="'''Correct!''' Thyrotrophin releasing hormone (TRH) from the hypothalamus stimulates the release of thyroid stimulating hormone (TSH) from the anterior pituitary which in turn stimulates the release of thyroid hormone form the thyroid gland. Somatostatin inhibits the release of TSH. [[Thyroid_Gland_- Anatomy & Physiology#Regulationn|WikiVet Article: Thyroid physiology]]"
-feedback4="'''Incorrect.''' Be careful not to confuse thyroid stimulating hormone(TSH) (secreted from the anterior pituitary to target the thyroid gland) with TRH (secreted from the hypothalamus to target the anterior pituitary's thyrotrophic cells). Also Growth Hormone does not inhibit TSH secretion. The correct stimulatory/inhibitory combination is Thyrotrophin Releasing Hormone / Somatostatin. [[Thyroid_Gland_-_Anatomy_%26_Physiology#Regulation|WikiVet Article: Thyroid physiology]]"
+feedback4="'''Incorrect.''' Be careful not to confuse thyroid stimulating hormone(TSH) (secreted from the anterior pituitary to target the thyroid gland) with TRH (secreted from the hypothalamus to target the anterior pituitary's thyrotrophic cells). Also Growth Hormone does not inhibit TSH secretion. The correct stimulatory/inhibitory combination is Thyrotrophin Releasing Hormone / Somatostatin. [[Thyroid_Gland_- Anatomy & Physiology#Regulation|WikiVet Article: Thyroid physiology]]"
-feedback3="'''Incorrect.''' Thyrotrophin releasing hormone does stimulate the secretion of thyroid stimulating hormone (TSH), however dopamine does not inhibit TSH secretion. Dopamine inhibits prolactin secretion from the anterior pituitary. The correct stimulatory/inhibitory combination is Thyrotrophin Releasing Hormone / Somatostatin. [[Thyroid_Gland_-_Anatomy_%26_Physiology#Regulation|WikiVet Article: Thyroid physiology]]"
+feedback3="'''Incorrect.''' Thyrotrophin releasing hormone does stimulate the secretion of thyroid stimulating hormone (TSH), however dopamine does not inhibit TSH secretion. Dopamine inhibits prolactin secretion from the anterior pituitary. The correct stimulatory/inhibitory combination is Thyrotrophin Releasing Hormone / Somatostatin. [[Thyroid_Gland_- Anatomy & Physiology#Regulation|WikiVet Article: Thyroid physiology]]"
-feedback1="'''Incorrect.''' Adrenocorticotrophic hormone is secreted form the anterior pituitary gland and it stimulates secretion of glucocorticoids from the adrenal cortex, it has no effect on the thyroid gland. Thyrotrophin Releasing Hormone does not inhibit the secretion of Thyroid Stimulating Hormone (TSH), it stimulates it. The correct stimulatory/inhibitory combination is Thyrotrophin Releasing Hormone / Somatostatin. [[Thyroid_Gland_-_Anatomy_%26_Physiology#Regulation|WikiVet Article: Thyroid physiology]]"
+feedback1="'''Incorrect.''' Adrenocorticotrophic hormone is secreted form the anterior pituitary gland and it stimulates secretion of glucocorticoids from the adrenal cortex, it has no effect on the thyroid gland. Thyrotrophin Releasing Hormone does not inhibit the secretion of Thyroid Stimulating Hormone (TSH), it stimulates it. The correct stimulatory/inhibitory combination is Thyrotrophin Releasing Hormone / Somatostatin. [[Thyroid_Gland_- Anatomy & Physiology#Regulation|WikiVet Article: Thyroid physiology]]"
-feedback2="'''Incorrect.''' Be careful not to confuse thyroid stimulating hormone (TSH) (secreted from the anterior pituitary to target the thyroid gland) with Thyrotrophin Releasing Hormone (TRH) (secreted from the hypothalamus to target the anterior pituitary's thyrotrophic cells). Somatostatin is inhibitory though. The correct stimulatory/inhibitory combination is Thyrotrophin Releasing Hormone / Somatostatin. [[Thyroid_Gland_-_Anatomy_%26_Physiology#Regulation|WikiVet Article: Thyroid physiology]]"
+feedback2="'''Incorrect.''' Be careful not to confuse thyroid stimulating hormone (TSH) (secreted from the anterior pituitary to target the thyroid gland) with Thyrotrophin Releasing Hormone (TRH) (secreted from the hypothalamus to target the anterior pituitary's thyrotrophic cells). Somatostatin is inhibitory though. The correct stimulatory/inhibitory combination is Thyrotrophin Releasing Hormone / Somatostatin. [[Thyroid_Gland_- Anatomy & Physiology#Regulation|WikiVet Article: Thyroid physiology]]"
 image= "">
 </WikiQuiz>
@@ Line 111: / Line 111: @@
 choice3="Posterior pituitary nuclei"
 correctchoice="2"
-feedback2="'''Correct!''' Prolactin is produced in the lactotrophs of the anterior pituitary and it acts on mammary secretory epithelial cells to stimulate synthesis of milk proteins. [[Pituitary_Gland_-_Anatomy_%26_Physiology#Hormones_of_the_Anterior_Pituitary_Gland|WikiVet Article: Anterior pituitary hormones]]"
+feedback2="'''Correct!''' Prolactin is produced in the lactotrophs of the anterior pituitary and it acts on mammary secretory epithelial cells to stimulate synthesis of milk proteins. [[Pituitary_Gland_- Anatomy & Physiology#Hormones_of_the_Anterior_Pituitary_Gland|WikiVet Article: Anterior pituitary hormones]]"
-feedback5="'''Incorrect.''' The somatotrophs of the anterior pituitary produce growth hormone. Prolactin is produced in the lactotrophs of the anterior pituitary. [[Pituitary_Gland_-_Anatomy_%26_Physiology#Hormones_of_the_Anterior_Pituitary_Gland|WikiVet Article: Anterior pituitary hormones]]"
+feedback5="'''Incorrect.''' The somatotrophs of the anterior pituitary produce growth hormone. Prolactin is produced in the lactotrophs of the anterior pituitary. [[Pituitary_Gland_- Anatomy & Physiology#Hormones_of_the_Anterior_Pituitary_Gland|WikiVet Article: Anterior pituitary hormones]]"
-feedback1="'''Incorrect.''' Prolactin acts on mammary secretory epithelial cells to stimulate synthesis of milk proteins. Prolactin is produced in the lactotrophs of the anterior pituitary. [[Pituitary_Gland_-_Anatomy_%26_Physiology#Hormones_of_the_Anterior_Pituitary_Gland|WikiVet Article: Anterior pituitary hormones]]"
+feedback1="'''Incorrect.''' Prolactin acts on mammary secretory epithelial cells to stimulate synthesis of milk proteins. Prolactin is produced in the lactotrophs of the anterior pituitary. [[Pituitary_Gland_- Anatomy & Physiology#Hormones_of_the_Anterior_Pituitary_Gland|WikiVet Article: Anterior pituitary hormones]]"
-feedback4="'''Incorrect.''' The placenta produces many hormones including prostaglandin F2α, progesterone, oestrogen and placental lactogen. Prolactin is produced in the lactotrophs of the anterior pituitary. [[Pituitary_Gland_-_Anatomy_%26_Physiology#Hormones_of_the_Anterior_Pituitary_Gland|WikiVet Article: Anterior pituitary hormones]]"
+feedback4="'''Incorrect.''' The placenta produces many hormones including prostaglandin F2α, progesterone, oestrogen and placental lactogen. Prolactin is produced in the lactotrophs of the anterior pituitary. [[Pituitary_Gland_- Anatomy & Physiology#Hormones_of_the_Anterior_Pituitary_Gland|WikiVet Article: Anterior pituitary hormones]]"
-feedback3="'''Incorrect.''' The posterior pituitary produces antidiuretic hormone (ADH) and oxytocin. Prolactin is produced in the lactotrophs of the anterior pituitary. [[Pituitary_Gland_-_Anatomy_%26_Physiology#Hormones_of_the_Anterior_Pituitary_Gland|WikiVet Article: Anterior pituitary hormones]]"
+feedback3="'''Incorrect.''' The posterior pituitary produces antidiuretic hormone (ADH) and oxytocin. Prolactin is produced in the lactotrophs of the anterior pituitary. [[Pituitary_Gland_- Anatomy & Physiology#Hormones_of_the_Anterior_Pituitary_Gland|WikiVet Article: Anterior pituitary hormones]]"
 image= "">
 </WikiQuiz>
@@ Line 144: / Line 144: @@
 choice5="Somatostatin, Dopamine and Oxytocin"
 correctchoice="4"
-feedback4="'''Correct!''' Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary_Gland_-_Anatomy_%26_Physiology#Posterior_Pituitary_Gland|WikiVet Article: Pituitary gland]]"
+feedback4="'''Correct!''' Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary_Gland_- Anatomy & Physiology#Posterior_Pituitary_Gland|WikiVet Article: Pituitary gland]]"
-feedback1="'''Incorrect.''' Prolactin is secreted by the anterior pituitary and dopamine is secreted by the hypothalamus. Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary_Gland_-_Anatomy_%26_Physiology#Posterior_Pituitary_Gland|WikiVet Article: Pituitary gland]]"
+feedback1="'''Incorrect.''' Prolactin is secreted by the anterior pituitary and dopamine is secreted by the hypothalamus. Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary_Gland_- Anatomy & Physiology#Posterior_Pituitary_Gland|WikiVet Article: Pituitary gland]]"
-feedback3="'''Incorrect.''' Prolactin is secreted by the anterior pituitary and somatostatin is secreted by the hypothalamus. Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary_Gland_-_Anatomy_%26_Physiology#Posterior_Pituitary_Gland|WikiVet Article: Pituitary gland]]"
+feedback3="'''Incorrect.''' Prolactin is secreted by the anterior pituitary and somatostatin is secreted by the hypothalamus. Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary_Gland_- Anatomy & Physiology#Posterior_Pituitary_Gland|WikiVet Article: Pituitary gland]]"
-feedback2="'''Incorrect.''' Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. Dopamine is synthesised in several areas of the brain, including the hypothalamus but is not secreted by the posterior pituitary. [[Pituitary_Gland_-_Anatomy_%26_Physiology#Posterior_Pituitary_Gland|WikiVet Article: Pituitary gland]]"
+feedback2="'''Incorrect.''' Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. Dopamine is synthesised in several areas of the brain, including the hypothalamus but is not secreted by the posterior pituitary. [[Pituitary_Gland_- Anatomy & Physiology#Posterior_Pituitary_Gland|WikiVet Article: Pituitary gland]]"
-feedback5="'''Incorrect.''' Somatostatin is secreted by the hypothalamus, dopamine is synthesised in several areas of the brain, including the hypothalamus but is not secreted by the posterior pituitary. Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary_Gland_-_Anatomy_%26_Physiology#Posterior_Pituitary_Gland|WikiVet Article: Pituitary gland]]"
+feedback5="'''Incorrect.''' Somatostatin is secreted by the hypothalamus, dopamine is synthesised in several areas of the brain, including the hypothalamus but is not secreted by the posterior pituitary. Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary_Gland_- Anatomy & Physiology#Posterior_Pituitary_Gland|WikiVet Article: Pituitary gland]]"
 image= "">
 </WikiQuiz>

@@ Line 31: / Line 31: @@
 choice2="Kidney"
 correctchoice="4"
-feedback4="'''Correct!''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
+feedback4="'''Correct!''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
-feedback5="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
+feedback5="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
-feedback3="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
+feedback3="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
-feedback1="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
+feedback1="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
-feedback2="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
+feedback2="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
 image= "">
 </WikiQuiz>
@@ Line 47: / Line 47: @@
 choice1="Insulin"
 correctchoice="2"
-feedback2="'''Correct!''' Parathyroid hormone stimulates the formation of active vitamin D3 (calcitriol) and inhibits the formation of inactive vitamin D3, (24,25 (OH)2 D3). The release of parathyroid hormone is inhibited by an increase in calcitriol and blood calcium levels (an example of negative feedback). [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]."
+feedback2="'''Correct!''' Parathyroid hormone stimulates the formation of active vitamin D3 (calcitriol) and inhibits the formation of inactive vitamin D3, (24,25 (OH)2 D3). The release of parathyroid hormone is inhibited by an increase in calcitriol and blood calcium levels (an example of negative feedback). [[Calcium#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]."
-feedback4="'''Incorrect.''' Thyroxine is not related to the regulation of the amount of active vitamin D3 in the body. Parathyroid hormone stimulates the formation of active vitamin D3 (calcitriol) and inhibits the formation of inactive vitamin D3, (24,25 (OH)2 D3). The release of parathyroid hormone is inhibited by an increase in calcitriol and blood calcium levels (an example of negative feedback). [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
+feedback4="'''Incorrect.''' Thyroxine is not related to the regulation of the amount of active vitamin D3 in the body. Parathyroid hormone stimulates the formation of active vitamin D3 (calcitriol) and inhibits the formation of inactive vitamin D3, (24,25 (OH)2 D3). The release of parathyroid hormone is inhibited by an increase in calcitriol and blood calcium levels (an example of negative feedback). [[Calcium#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
-feedback3="'''Incorrect.''' Triiodothyronine is not related to the regulation of the amount of active vitamin D3 in the body. Parathyroid hormone stimulates the formation of active vitamin D3 (calcitriol) and inhibits the formation of inactive vitamin D3, (24,25 (OH)2 D3). The release of parathyroid hormone is inhibited by an increase in calcitriol and blood calcium levels (an example of negative feedback). [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
+feedback3="'''Incorrect.''' Triiodothyronine is not related to the regulation of the amount of active vitamin D3 in the body. Parathyroid hormone stimulates the formation of active vitamin D3 (calcitriol) and inhibits the formation of inactive vitamin D3, (24,25 (OH)2 D3). The release of parathyroid hormone is inhibited by an increase in calcitriol and blood calcium levels (an example of negative feedback). [[Calcium#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
-feedback5="'''Incorrect.''' Epinephrine is not related to the regulation of the amount of active vitamin D3 in the body. Parathyroid hormone stimulates the formation of active vitamin D3 (calcitriol) and inhibits the formation of inactive vitamin D3, (24,25 (OH)2 D3). The release of parathyroid hormone is inhibited by an increase in calcitriol and blood calcium levels (an example of negative feedback). [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
+feedback5="'''Incorrect.''' Epinephrine is not related to the regulation of the amount of active vitamin D3 in the body. Parathyroid hormone stimulates the formation of active vitamin D3 (calcitriol) and inhibits the formation of inactive vitamin D3, (24,25 (OH)2 D3). The release of parathyroid hormone is inhibited by an increase in calcitriol and blood calcium levels (an example of negative feedback). [[Calcium#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
-feedback1="'''Incorrect.''' Insulin is not related to the regulation of the amount of active vitamin D3 in the body. Parathyroid hormone stimulates the formation of active vitamin D3 (calcitriol) and inhibits the formation of inactive vitamin D3, (24,25 (OH)2 D3). The release of parathyroid hormone is inhibited by an increase in calcitriol and blood calcium levels (an example of negative feedback). [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
+feedback1="'''Incorrect.''' Insulin is not related to the regulation of the amount of active vitamin D3 in the body. Parathyroid hormone stimulates the formation of active vitamin D3 (calcitriol) and inhibits the formation of inactive vitamin D3, (24,25 (OH)2 D3). The release of parathyroid hormone is inhibited by an increase in calcitriol and blood calcium levels (an example of negative feedback). [[Calcium#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]]"
 image= "">
 </WikiQuiz>
@@ Line 95: / Line 95: @@
 choice5="It decreases blood calcium by increasing renal excretion of calcium."
 correctchoice="4"
-feedback4="'''Correct!''' Calcitonin decreases blood calcium by decreasing osteolysis and increasing osteogenesis. [[Calcium Homeostasis - Anatomy & Physiology#Calcitonin |WikiVet Article: Calcium homeostasis]]"
+feedback4="'''Correct!''' Calcitonin decreases blood calcium by decreasing osteolysis and increasing osteogenesis. [[Calcium#Calcitonin |WikiVet Article: Calcium homeostasis]]"
-feedback2="'''Incorrect.''' Decreasing osteolysis and increasing osteogenesis will decrease, not increase blood calcium. Calcitonin decreases blood calcium by decreasing osteolysis and increasing osteogenesis. [[Calcium Homeostasis - Anatomy & Physiology#Calcitonin|WikiVet Article: Calcium homeostasis]]"
+feedback2="'''Incorrect.''' Decreasing osteolysis and increasing osteogenesis will decrease, not increase blood calcium. Calcitonin decreases blood calcium by decreasing osteolysis and increasing osteogenesis. [[Calcium#Calcitonin|WikiVet Article: Calcium homeostasis]]"
-feedback1="'''Incorrect.''' Increased osteolysis and decreased osteogenesis will increase blood calcium. Calcitonin decreases blood calcium by decreasing osteolysis and increasing osteogenesis. [[Calcium Homeostasis - Anatomy & Physiology#Calcitonin |WikiVet Article: Calcium homeostasis]]"
+feedback1="'''Incorrect.''' Increased osteolysis and decreased osteogenesis will increase blood calcium. Calcitonin decreases blood calcium by decreasing osteolysis and increasing osteogenesis. [[Calcium#Calcitonin |WikiVet Article: Calcium homeostasis]]"
-feedback3="'''Incorrect.''' Calcitonin decreases blood calcium by decreasing osteolysis and increasing osteogenesis. PTH causes increased blood calcium by increasing osteolysis and decreasing osteogenesis. [[Calcium Homeostasis - Anatomy & Physiology#Calcitonin|WikiVet Article: Calcium homeostasis]]"
+feedback3="'''Incorrect.''' Calcitonin decreases blood calcium by decreasing osteolysis and increasing osteogenesis. PTH causes increased blood calcium by increasing osteolysis and decreasing osteogenesis. [[Calcium#Calcitonin|WikiVet Article: Calcium homeostasis]]"
-feedback5="'''Incorrect.''' Calcitonin has its effects on bone, not the kidney. Calcitonin decreases blood calcium by decreasing osteolysis and increasing osteogenesis. [[Calcium Homeostasis - Anatomy & Physiology#Calcitonin |WikiVet Article: Calcium homeostasis]]"
+feedback5="'''Incorrect.''' Calcitonin has its effects on bone, not the kidney. Calcitonin decreases blood calcium by decreasing osteolysis and increasing osteogenesis. [[Calcium#Calcitonin |WikiVet Article: Calcium homeostasis]]"
 image= "">
 </WikiQuiz>
@@ Line 208: / Line 208: @@
 choice4="PTH increases renal excretion of calcitriol."
 correctchoice="3"
-feedback3="'''Correct!''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: Calcium homeostasis]]"
+feedback3="'''Correct!''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium|WikiVet Article: Calcium homeostasis]]"
-feedback5="'''Incorrect.''' The 25- hydroxylation reaction in the liver is the unregulated step that produces calcidiol. PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: Calcium homeostasis]]"
+feedback5="'''Incorrect.''' The 25- hydroxylation reaction in the liver is the unregulated step that produces calcidiol. PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium|WikiVet Article: Calcium homeostasis]]"
-feedback2="'''Incorrect.''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: Calcium homeostasis]]"
+feedback2="'''Incorrect.''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium|WikiVet Article: Calcium homeostasis]]"
-feedback1="'''Incorrect.''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: Calcium homeostasis]]"
+feedback1="'''Incorrect.''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium|WikiVet Article: Calcium homeostasis]]"
-feedback4="'''Incorrect.''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: Calcium homeostasis]]"
+feedback4="'''Incorrect.''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium|WikiVet Article: Calcium homeostasis]]"
 image= "">
 </WikiQuiz>

@@ Line 63: / Line 63: @@
 choice3="Sodium, chloride, potassium and water excretion."
 correctchoice="2"
-feedback2="'''Correct!''' Renin increases aldosterone secretion from the zona glomerulosa. Aldosterone acts to increase the reabsorption of sodium by cells in the collecting ducts of the kidney. Sodium ions are exchanged for hydrogen and potassium ions, leading to decreased sodium and increased potassium excretion. Water follows sodium, blood volume increases, and this increases blood pressure and glomerular filtration rate. [[Important Hormonal Regulators of the Kidney - Anatomy & Physiology#The Renin Angiotensin Aldosterone System .28RAAS.29|WikiVet Article: RAAS. ]]"
+feedback2="'''Correct!''' Renin increases aldosterone secretion from the zona glomerulosa. Aldosterone acts to increase the reabsorption of sodium by cells in the collecting ducts of the kidney. Sodium ions are exchanged for hydrogen and potassium ions, leading to decreased sodium and increased potassium excretion. Water follows sodium, blood volume increases, and this increases blood pressure and glomerular filtration rate. [[Renin Angiotensin Aldosterone System|WikiVet Article: RAAS. ]]"
-feedback1="'''Incorrect.''' Renin increases aldosterone secretion from the zona glomerulosa. Aldosterone acts to increase the reabsorption of sodium by cells in the collecting ducts of the kidney. Sodium ions are exchanged for hydrogen and potassium ions, leading to decreased sodium and increased potassium excretion. Water follows sodium, blood volume increases, and this increases blood pressure and glomerular filtration rate. [[Important Hormonal Regulators of the Kidney - Anatomy & Physiology#The Renin Angiotensin Aldosterone System .28RAAS.29|WikiVet Article: RAAS]]."
+feedback1="'''Incorrect.''' Renin increases aldosterone secretion from the zona glomerulosa. Aldosterone acts to increase the reabsorption of sodium by cells in the collecting ducts of the kidney. Sodium ions are exchanged for hydrogen and potassium ions, leading to decreased sodium and increased potassium excretion. Water follows sodium, blood volume increases, and this increases blood pressure and glomerular filtration rate. [[Renin Angiotensin Aldosterone System|WikiVet Article: RAAS]]."
-feedback4="'''Incorrect.''' Renin increases aldosterone secretion from the zona glomerulosa. Aldosterone acts to increase the reabsorption of sodium by cells in the collecting ducts of the kidney. Sodium ions are exchanged for hydrogen and potassium ions, leading to decreased sodium and increased potassium excretion. Water follows sodium, blood volume increases, and this increases blood pressure and glomerular filtration rate. [[Important Hormonal Regulators of the Kidney - Anatomy & Physiology#The Renin Angiotensin Aldosterone System .28RAAS.29|WikiVet Article: RAAS]]."
+feedback4="'''Incorrect.''' Renin increases aldosterone secretion from the zona glomerulosa. Aldosterone acts to increase the reabsorption of sodium by cells in the collecting ducts of the kidney. Sodium ions are exchanged for hydrogen and potassium ions, leading to decreased sodium and increased potassium excretion. Water follows sodium, blood volume increases, and this increases blood pressure and glomerular filtration rate. [[Renin Angiotensin Aldosterone System|WikiVet Article: RAAS]]."
-feedback5="'''Incorrect.''' Renin increases aldosterone secretion from the zona glomerulosa. Aldosterone acts to increase the reabsorption of sodium by cells in the collecting ducts of the kidney. Sodium ions are exchanged for hydrogen and potassium ions, leading to decreased sodium and increased potassium excretion. Water follows sodium, blood volume increases, and this increases blood pressure and glomerular filtration rate. [[Important Hormonal Regulators of the Kidney - Anatomy & Physiology#The Renin Angiotensin Aldosterone System .28RAAS.29|WikiVet Article: RAAS]]."
+feedback5="'''Incorrect.''' Renin increases aldosterone secretion from the zona glomerulosa. Aldosterone acts to increase the reabsorption of sodium by cells in the collecting ducts of the kidney. Sodium ions are exchanged for hydrogen and potassium ions, leading to decreased sodium and increased potassium excretion. Water follows sodium, blood volume increases, and this increases blood pressure and glomerular filtration rate. [[Renin Angiotensin Aldosterone System|WikiVet Article: RAAS]]."
-feedback3="'''Incorrect.''' Renin increases aldosterone secretion from the zona glomerulosa. Aldosterone acts to increase the reabsorption of sodium by cells in the collecting ducts of the kidney. Sodium ions are exchanged for hydrogen and potassium ions, leading to decreased sodium and increased potassium excretion. Water follows sodium, blood volume increases, and this increases blood pressure and glomerular filtration rate. [[Important Hormonal Regulators of the Kidney - Anatomy & Physiology#The Renin Angiotensin Aldosterone System .28RAAS.29|WikiVet Article: RAAS]]."
+feedback3="'''Incorrect.''' Renin increases aldosterone secretion from the zona glomerulosa. Aldosterone acts to increase the reabsorption of sodium by cells in the collecting ducts of the kidney. Sodium ions are exchanged for hydrogen and potassium ions, leading to decreased sodium and increased potassium excretion. Water follows sodium, blood volume increases, and this increases blood pressure and glomerular filtration rate. [[Renin Angiotensin Aldosterone System|WikiVet Article: RAAS]]."
 image= "">
 </WikiQuiz>

@@ Line 127: / Line 127: @@
 choice4="GLUT 4"
 correctchoice="5"
-feedback5="'''Correct!''' GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and Absorption|WikiVet Article: Carbohydrate digestion and absorption]]"
+feedback5="'''Correct!''' GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine Overview - Anatomy & Physiology#Carbohydrate Digestion and Absorption|WikiVet Article: Carbohydrate digestion and absorption]]"
-feedback3="'''Incorrect.''' The glucose/Na+ symport is responsible for the uptake of glucose from the intestinal lumen into intestinal cells. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and Absorption|WikiVet Article: Carbohydrate digestion and absorption]]"
+feedback3="'''Incorrect.''' The glucose/Na+ symport is responsible for the uptake of glucose from the intestinal lumen into intestinal cells. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine Overview - Anatomy & Physiology#Carbohydrate Digestion and Absorption|WikiVet Article: Carbohydrate digestion and absorption]]"
-feedback2="'''Incorrect.''' The Na+/K+ ATPase is responsible for pumping sodium ions into the blood in order to maintain a low concentration of sodium inside the intestinal cells. This is important as the action of the glucose/Na+ symport depends upon their being a lower concentration of sodium inside the intestinal cells than in the gut lumen. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and Absorption|WikiVet Article: Carbohydrate digestion and absorption]]"
+feedback2="'''Incorrect.''' The Na+/K+ ATPase is responsible for pumping sodium ions into the blood in order to maintain a low concentration of sodium inside the intestinal cells. This is important as the action of the glucose/Na+ symport depends upon their being a lower concentration of sodium inside the intestinal cells than in the gut lumen. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine Overview - Anatomy & Physiology#Carbohydrate Digestion and Absorption|WikiVet Article: Carbohydrate digestion and absorption]]"
-feedback1="'''Incorrect.''' γ Glutamyl transferase spans the enterocyte membrane and combines glutathione from the inside of the cell with a di-,tri- or oligo-peptide from the intestinal lumen forming a γ-glu-aa complex which is transported into the cell. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and Absorption |WikiVet Article: Carbohydrate digestion and absorption]]"
+feedback1="'''Incorrect.''' γ Glutamyl transferase spans the enterocyte membrane and combines glutathione from the inside of the cell with a di-,tri- or oligo-peptide from the intestinal lumen forming a γ-glu-aa complex which is transported into the cell. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine Overview - Anatomy & Physiology#Carbohydrate Digestion and Absorption |WikiVet Article: Carbohydrate digestion and absorption]]"
-feedback4="'''Incorrect.''' GLUT 4 transporters are used for uptake of glucose into muscle and adipose tissue cells. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and AbsorptionPancreas - Anatomy & Physiology|WikiVet Article: Carbohydrate digestion and absorption]]"
+feedback4="'''Incorrect.''' GLUT 4 transporters are used for uptake of glucose into muscle and adipose tissue cells. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine Overview - Anatomy & Physiology#Carbohydrate Digestion and AbsorptionPancreas - Anatomy & Physiology|WikiVet Article: Carbohydrate digestion and absorption]]"
 image= "">
 </WikiQuiz>

@@ Line 15: / Line 15: @@
 choice2="Glucocorticoids, insulin and thyroxine (T4)"
 correctchoice="5"
-feedback5="'''Correct!''' Growth hormone is the main regulator of IGF-I production in the liver. Insulin and oestradiol are stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]."
+feedback5="'''Correct!''' Growth hormone is the main regulator of IGF-I production in the liver. Insulin and oestradiol are stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]"
-feedback4="'''Incorrect.''' Thyroxine (T4) does not directly affect IGF-I production. However growth hormone is the main regulator of IGF-I production in the liver and insulin is stimulatory in other tissues. The missing hormone is oestradiol which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]."
+feedback4="'''Incorrect.''' Thyroxine (T4) does not directly affect IGF-I production. However growth hormone is the main regulator of IGF-I production in the liver and insulin is stimulatory in other tissues. The missing hormone is oestradiol which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]"
-feedback3="'''Incorrect.''' Growth hormone is the main regulator of IGF-I production in the liver and insulin is stimulatory in other tissues. Glucocorticoids are inhibitory in other tissues. The missing hormone is oestradiol which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]."
+feedback3="'''Incorrect.''' Growth hormone is the main regulator of IGF-I production in the liver and insulin is stimulatory in other tissues. Glucocorticoids are inhibitory in other tissues. The missing hormone is oestradiol which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]"
-feedback1="'''Incorrect.''' Growth hormone is the main regulator of IGF-I production in the liver and oestradiol is stimulatory in other tissues. Glucocorticoids are inhibitory in other tissues. The missing hormone is insulin which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]."
+feedback1="'''Incorrect.''' Growth hormone is the main regulator of IGF-I production in the liver and oestradiol is stimulatory in other tissues. Glucocorticoids are inhibitory in other tissues. The missing hormone is insulin which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]"
-feedback2="'''Incorrect.''' Insulin is stimulatory in many tissues and glucocorticoids are inhibitory in several tissues. Thyroxine (T4) does not directly affect IGF-I production.The missing hormones are growth hormone which is the main regulator of IGF-I production in the liver and oestradiol which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]."
+feedback2="'''Incorrect.''' Insulin is stimulatory in many tissues and glucocorticoids are inhibitory in several tissues. Thyroxine (T4) does not directly affect IGF-I production.The missing hormones are growth hormone which is the main regulator of IGF-I production in the liver and oestradiol which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]"
 image= "">
 </WikiQuiz>
@@ Line 31: / Line 31: @@
 choice2="Kidney"
 correctchoice="4"
-feedback4="'''Correct!''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: active vitamin D synthesis]]."
+feedback4="'''Correct!''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]."
-feedback5="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: active vitamin D synthesis]]."
+feedback5="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]."
-feedback3="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: active vitamin D synthesis]]."
+feedback3="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]."
-feedback1="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: active vitamin D synthesis]]."
+feedback1="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]."
-feedback2="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: active vitamin D synthesis]]."
+feedback2="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]."
 image= "">
 </WikiQuiz>