Modified Transudate - Revision history

Fiorecastro at 17:56, 4 January 2023

2023-01-04T17:56:17Z

Bara at 13:28, 18 July 2012

2012-07-18T13:28:53Z

Bara at 17:05, 24 September 2011

2011-09-24T17:05:49Z

Bara: Text replace - "Category:To_Do_-_James" to ""

2010-10-09T22:30:03Z

Text replace - "Category:To_Do_-_James" to ""

Suzannah.stacey: /* Description */

2010-10-09T16:04:21Z

Description

Bara at 21:43, 2 October 2010

2010-10-02T21:43:13Z

Bara at 15:14, 9 September 2010

2010-09-09T15:14:19Z

Bara: Text replace - "[[Lymphocytes|" to "[[Lymphocytes - Introduction|"

2010-08-18T12:42:03Z

Text replace - "[[Lymphocytes|" to "[[Lymphocytes - Introduction|"

Bara at 12:12, 29 July 2010

2010-07-29T12:12:18Z

Bara: /* Description */

2010-07-27T10:28:12Z

Description

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-{{OpenPagesTop}}
 ==Introduction==
 Modified transudates are a type of effusion whose cell count and protein content are intermediate between a [[Transudate|transudate]] and an [[Exudate|exudate]].  In some cases, modified transudates may represent a transitional stage before the development of an exudate (as with uroabdomen, which begins as a modified transudate but develops into a chemical exudative peritonitis over time).  Modified transudates arise as a result of disruptions to the endothelium or imbalances in the [[Blood Pressure Physiology#Capillary Fluid Shift Mechanism |'''Starling forces''']].  Common causes include:
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 {{review}}
-{{OpenPages}}
+==Webinars==
 [[Category:Effusions]]
 [[Category:Expert_Review]]

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 ==Introduction==
 Modified transudates are a type of effusion whose cell count and protein content are intermediate between a [[Transudate|transudate]] and an [[Exudate|exudate]].  In some cases, modified transudates may represent a transitional stage before the development of an exudate (as with uroabdomen, which begins as a modified transudate but develops into a chemical exudative peritonitis over time).  Modified transudates arise as a result of disruptions to the endothelium or imbalances in the [[Blood Pressure Physiology#Capillary Fluid Shift Mechanism |'''Starling forces''']].  Common causes include:
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 }}
 [[Category:Effusions]]
 [[Category:Expert_Review]]

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-{{review}}
+==Introduction==
 Modified transudates are a type of effusion whose cell count and protein content are intermediate between a [[Transudate|transudate]] and an [[Exudate|exudate]].  In some cases, modified transudates may represent a transitional stage before the development of an exudate (as with uroabdomen, which begins as a modified transudate but develops into a chemical exudative peritonitis over time).  Modified transudates arise as a result of disruptions to the endothelium or imbalances in the [[Blood Pressure Physiology#Capillary Fluid Shift Mechanism |'''Starling forces''']].  Common causes include:
 *'''Increased vascular hydrostatic pressure''' resulting from inappropriate activation of the renin-angiotensin-aldosterone system (RAAS) in '''portal hypertension''' or '''congestive heart failure'''.
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 The presence of neoplastic cells in an effusion should prompt further investigation through diagnostic imaging or exploratory surgery.
-==Literature Search==
+{{Learning
-[[File:CABI logo.jpg|left|90px]]
+|literature search = [http://www.cabdirect.org/search.html?q=ab%3A%28%22Modified+Transudate%22%29 Modified transudate publications]
 [[Category:Effusions]]
 [[Category:Expert_Review]]

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 [[Category:Effusions]]
-[[Category:To_Do_-_James]]
 [[Category:Expert_Review]]

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 ==Description==
-Modified transudates are a type of effusion whose cell count and protein content are intermediate between a [[Transudate|transudate]] and an [[Exudate|exudate]].  In some cases, modified transudates may represent a transitional stage before the development of an exudate (as with uroabdomen, which begins as a modified transudate but develops into a chemical exudative peritonitis over time).  Modified transudates arise as a result of disruptions to the endothelium or imbalances in the [[Blood Pressure & Blood Flow Control - Anatomy & Physiology#Capillary Fluid Shift Mechanism |'''Starling forces''']].  Common causes include:
+Modified transudates are a type of effusion whose cell count and protein content are intermediate between a [[Transudate|transudate]] and an [[Exudate|exudate]].  In some cases, modified transudates may represent a transitional stage before the development of an exudate (as with uroabdomen, which begins as a modified transudate but develops into a chemical exudative peritonitis over time).  Modified transudates arise as a result of disruptions to the endothelium or imbalances in the [[Blood Pressure Physiology#Capillary Fluid Shift Mechanism |'''Starling forces''']].  Common causes include:
 *'''Increased vascular hydrostatic pressure''' resulting from inappropriate activation of the renin-angiotensin-aldosterone system (RAAS) in '''portal hypertension''' or '''congestive heart failure'''.
 *'''Vasculitis''', caused by '''[[Feline Infectious Peritonitis|feline infectious peritonitis virus]]''' (FIPV) or '''neoplasia'''.  ([[Feline Infectious Peritonitis|Further discussion of the effusion encountered with FIP.]])

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 |-
 | Nucleated cells
-| 0.3 - 5.5 x10e9/L (up to 7 x 10e9/L), of which the majority are [[Mesothelial cells|mesothelial cells]], [[Macrophages|macrophages]], non-degenerate neutrophils and small [[Lymphocytes|lymphocytes]]
+| 0.3 - 5.5 x10e9/L (up to 7 x 10e9/L), of which the majority are [[Mesothelial cells|mesothelial cells]], [[Macrophages|macrophages]], non-degenerate neutrophils and small [[Lymphocytes - Introduction|lymphocytes]]
 |}

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 Modified transudates are a type of effusion whose cell count and protein content are intermediate between a [[Transudate|transudate]] and an [[Exudate|exudate]].  In some cases, modified transudates may represent a transitional stage before the development of an exudate (as with uroabdomen, which begins as a modified transudate but develops into a chemical exudative peritonitis over time).  Modified transudates arise as a result of disruptions to the endothelium or imbalances in the [[Control of Blood Pressure & Blood Flow - Anatomy & Physiology#Capillary Fluid Shift Mechanism |'''Starling forces''']].  Common causes include:
 *'''Increased vascular hydrostatic pressure''' resulting from inappropriate activation of the renin-angiotensin-aldosterone system (RAAS) in '''portal hypertension''' or '''congestive heart failure'''.
-*'''Vasculitis''', caused by '''[[Feline Infectious Peritonitis|feline infectious peritonitis virus]]''' (FIPV) or '''neoplasia'''.  For further discussion of the effusion encountered with FIP, see [[Feline Infectious Peritonitis|here]].
+*'''Vasculitis''', caused by '''[[Feline Infectious Peritonitis|feline infectious peritonitis virus]]''' (FIPV) or '''neoplasia'''.  ([[Feline Infectious Peritonitis|Further discussion of the effusion encountered with FIP.]])
 *'''Strangulation of abdominal or thoracic organs''' may lead to the production of a modified transudate (which will develop into an exudate if untreated).  This may occur with '''lung lobe torsion''', '''torsion of the spleen''' or of a [[Liver Torsion|liver lobe]] or if part of the liver becomes strangulated within a [[Diaphragmatic Rupture|'''diaphragmatic rupture''']].
 *'''[[Chylous Effusion|Chylous effusions''']] are sometimes considered to be a type of modified transudate.