Strongyloides - Revision history

Xxaoifedxx: /* Clinical Signs */

2019-05-31T14:10:40Z

Clinical Signs

Xxaoifedxx: /* Diagnosis */

2019-05-31T14:10:23Z

Diagnosis

Xxaoifedxx: /* Clinical Signs */

2019-05-31T14:10:10Z

Clinical Signs

Xxaoifedxx: /* Pathology */

2019-05-31T14:09:57Z

Pathology

Xxaoifedxx: /* Transmission */

2019-05-31T14:09:43Z

Transmission

Xxaoifedxx: /* Treatment */

2019-05-31T14:09:28Z

Treatment

Xxaoifedxx: /* Transmission */

2019-05-31T14:07:33Z

Transmission

Xxaoifedxx: /* Overview */

2019-05-31T14:07:04Z

Overview

Xxaoifedxx: /* Pathogenicity */

2019-05-31T13:57:16Z

Pathogenicity

Vulpesferrilata: /* Life-Cycle */

2013-06-12T13:18:34Z

Life-Cycle

@@ Line 49: / Line 49: @@
 ====Life Cycle====
 The life cylce of ''S. papillosus'' is typical of [[:Category:Rhabditoidea|strongyloides]] species with both parasitic and free living cycles. As adults animals will become immune to infection the primary method of infection for young animals is through the milk of the dam, this still involves only females as males are only present in the environment as 'free-living' nematodes.
 [[Category:Rhabditoidea]][[Category:Horse Nematodes]][[Category:Sheep Nematodes]]
 [[Category:To_Do_-_Parasites]][[Category:To Do - Major]]

@@ Line 51: / Line 51: @@
 == Clinical Signs ==
 Not obvious. Slight unthriftiness. Maybe diarrhoea which is sometimes watery. Unkept coats.
 [[Category:Rhabditoidea]][[Category:Horse Nematodes]][[Category:Sheep Nematodes]]
 [[Category:To_Do_-_Parasites]][[Category:To Do - Major]]

@@ Line 49: / Line 49: @@
 ====Life Cycle====
 The life cylce of ''S. papillosus'' is typical of [[:Category:Rhabditoidea|strongyloides]] species with both parasitic and free living cycles. As adults animals will become immune to infection the primary method of infection for young animals is through the milk of the dam, this still involves only females as males are only present in the environment as 'free-living' nematodes.
 == Clinical Signs ==
 Not obvious. Slight unthriftiness. Maybe diarrhoea which is sometimes watery. Unkept coats.

@@ Line 49: / Line 49: @@
 ====Life Cycle====
 The life cylce of ''S. papillosus'' is typical of [[:Category:Rhabditoidea|strongyloides]] species with both parasitic and free living cycles. As adults animals will become immune to infection the primary method of infection for young animals is through the milk of the dam, this still involves only females as males are only present in the environment as 'free-living' nematodes.
 == Clinical Signs ==
 * 1st stage to skin - dermatitis

@@ Line 49: / Line 49: @@
 ====Life Cycle====
 The life cylce of ''S. papillosus'' is typical of [[:Category:Rhabditoidea|strongyloides]] species with both parasitic and free living cycles. As adults animals will become immune to infection the primary method of infection for young animals is through the milk of the dam, this still involves only females as males are only present in the environment as 'free-living' nematodes.
 == Pathology ==
 Adults burrow into mucosa of the proximal third part of the small intestine. Inflammation, erosion and oedema of epithelium occurs. Impairment of digestion and absorption also occurs.

@@ Line 55: / Line 55: @@
 Definite host is pigs.
 == Transmission ==
-  Transmammary, skin penetration, mucosa of oral cavity penetration (after ingestion)
+  Transmammary, skin penetration, mucosa of oral cavity penetration (after ingestion).
 == Pathology ==
 Adults burrow into mucosa of the proximal third part of the small intestine. Inflammation, erosion and oedema of epithelium occurs. Impairment of digestion and absorption also occurs.

@@ Line 38: / Line 38: @@
 ==== Pathogenicity ====
 Pathology is rarely seen in adult horses due to the development of immunity, though penetration of the skin by larvae may still cause irritation anddermatitis. Adults with larvae in beneath the skin and in the abdomen can act as carriers. In foals heavy infections can cause severe enteritis and diarrhoea. It should be noted that apparently healthy animals can still have high faecal egg counts.
 ==== Control ====
 The number and spread of ''S. westeri'' is best controlled by good hygeine such as the removal of faeces from pasture and stabling and the provision of clean, dry bedding. Foals are often treated with anthelmintics at 2 weeks old against ''S. westeri''.

@@ Line 34: / Line 34: @@
 The eggs are small and oval with a thin shell, they will often hatch in the large intestine so L1 larvae are expelled in the hosts faeces.
 ==== Life-Cycle ====
-''S. westeri'' has a typical [[:Category:Rhabditoidea|strongyloides]] life cycle, with both a free living and parasitic cycle. The free living cycle involves male and female worms reproducing sexually on the ground, this can occur for several generation with no parasitism taking place. The parasitic phase involves only female worms and occurs from ingesion of L£ larvae or their penetration through the hosts skin. Infection my also be transmissed vertically from the dam to the foal by larval migration to the mammary tissue and ingestion by the foal with the dams milk. Once in the host the larvae migrate to the small intestine where they tunnel into the epithelium at the base of the villi and moult to L4 and then adult females. Females produce eggs without sexual interaction that can develop to become both male and female larvae. The pre patent period is between 1 and 2 weeks dependant on the level of infection.
+''S. westeri'' has a typical [[:Category:Rhabditoidea|strongyloides]] life cycle, with both a free living and parasitic cycle. The free living cycle involves male and female worms reproducing sexually on the ground, this can occur for several generation with no parasitism taking place. The parasitic phase involves only female worms and occurs from ingesion of L3 larvae or their penetration through the hosts skin. Infection my also be transmissed vertically from the dam to the foal by larval migration to the mammary tissue and ingestion by the foal with the dams milk. Once in the host the larvae migrate to the small intestine where they tunnel into the epithelium at the base of the villi and moult to L4 and then adult females. Females produce eggs without sexual interaction that can develop to become both male and female larvae. The pre patent period is between 1 and 2 weeks dependant on the level of infection.
 ==== Pathogenicity ====
 Pathology is rarely seen in adult horses due to the development of immunity, though penetration of the skin by larvae may still cause irritation anddermatitis. Adults with larvae in beneath the skin and in the abdomen can act as carriers. In foals heavy infections can cause severe enteritis and diarrhoea. It should be noted that apparently healthy animals can still have high faecal egg counts.