Difference between revisions of "Essential Ion and Compound Balance and Homeostasis - Renal Flash Cards - Anatomy & Physiology"
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+ | '''Use the mouse to highlight the answers. They are written in white''' | ||
+ | |||
+ | <TABLE BORDER="2" BORDERCOLOR="#6600FF" CELLPADDING="2" CELLSPACING="2" WIDTH="80%"> | ||
+ | <TR> | ||
+ | <TD>'''Question'''</TD> | ||
+ | <TD>'''Answer'''</TD> | ||
+ | <TD>'''Link'''</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>What the effects of varying sodium concentration within the body?</TD> | ||
+ | <TD><FONT COLOR="#FFFFFF"> | ||
+ | *Too low | ||
+ | **Water and ECF volumes fall | ||
+ | **Sodium dependant transporters are disturbed | ||
+ | *Too High | ||
+ | **Water and ECF volume increases | ||
+ | **Sodium dependant transporters are disturbed </font></TD> | ||
+ | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#Sodium|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>Why is sodium not regulated on the basis of plasma concentration?</TD> | ||
+ | <TD><FONT COLOR="#FFFFFF"> | ||
+ | *Water follows sodium. Therefore volume notconcentration alter | ||
+ | *The hormonal controllers affect sodium concentration and ECF concentration | ||
+ | *ADH and the thirst response dilute any increase in sodium by increasing ECF volume</font></TD> | ||
+ | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#Regulation|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>What is "salt hunger"?</TD> | ||
+ | <TD><FONT COLOR="#FFFFFF"> | ||
+ | It's where an animal with low body salt concentrations actually craves salt</font></TD> | ||
+ | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#Salt Hunger|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>What are the three major sources of potassium?</TD> | ||
+ | <TD><FONT COLOR="#FFFFFF"> | ||
+ | * Diffusion from the small intestine | ||
+ | * Active transport from the colon | ||
+ | * Recovered from cellular breakdown</font></TD> | ||
+ | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#Sources|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>By which three methods is potassium homeostasis managed? Which of these make up the chronic and which the acute response</TD> | ||
+ | <TD><FONT COLOR="#FFFFFF"> | ||
+ | *Cellular translocation - Acute | ||
+ | *Renal excretion - 90% of chronic | ||
+ | *GI excretion - 10% of chronic</font></TD> | ||
+ | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#Methods of Control|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>Which two things control the acute response and how do they do it?</TD> | ||
+ | <TD><FONT COLOR="#FFFFFF"> | ||
+ | *Insulin | ||
+ | *Activation of beta2 adrenoreceptors | ||
+ | *Work by increasing the activity of Na+ / K+ ATPases causing sodium efflux and potassium influx</font></TD> | ||
+ | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>Which hormone is the main regulator of potassium??</TD> | ||
+ | <TD><FONT COLOR="#FFFFFF"> | ||
+ | Aldosterone</font></TD> | ||
+ | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#Potassium and Aldosterone|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>In which two regions of the nephron is H<sup>+</sup> secreted?</TD> | ||
+ | <TD><FONT COLOR="#FFFFFF"> | ||
+ | *Proximal tubule | ||
+ | *Collecting duct</font></TD> | ||
+ | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#Acid / Base|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>What role do buffers play with regard to excreting hydrogen ions?</TD> | ||
+ | <TD><FONT COLOR="#FFFFFF"> | ||
+ | Once all bicarbonate has been reabsorbed they combine with residual excess hydrogen allowing it to be excreted without the loss of bicarbonate and without making the urine very acidic.</font></TD> | ||
+ | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#Buffers of H+ in Urine|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>In ruminants fed low nitrogen diets more urea is reabsorbed. Why?</TD> | ||
+ | <TD><FONT COLOR="#FFFFFF"> | ||
+ | It is transported to the rumen and converted to microbial protein</font></TD> | ||
+ | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#Urea and Foregut Fermenters|Link]]</TD> | ||
+ | </TR> | ||
+ | </table> | ||
+ | |||
+ | |||
+ | |||
+ | [[Category:Nephron]][[Category:Urinary System Anatomy & Physiology Flashcards]] |
Latest revision as of 17:21, 21 June 2011
Use the mouse to highlight the answers. They are written in white
Question | Answer | Link |
What the effects of varying sodium concentration within the body? |
|
Link |
Why is sodium not regulated on the basis of plasma concentration? |
|
Link |
What is "salt hunger"? | It's where an animal with low body salt concentrations actually craves salt | Link |
What are the three major sources of potassium? |
|
Link |
By which three methods is potassium homeostasis managed? Which of these make up the chronic and which the acute response |
|
Link |
Which two things control the acute response and how do they do it? |
|
Link |
Which hormone is the main regulator of potassium?? | Aldosterone | Link |
In which two regions of the nephron is H+ secreted? |
|
Link |
What role do buffers play with regard to excreting hydrogen ions? | Once all bicarbonate has been reabsorbed they combine with residual excess hydrogen allowing it to be excreted without the loss of bicarbonate and without making the urine very acidic. | Link |
In ruminants fed low nitrogen diets more urea is reabsorbed. Why? | It is transported to the rumen and converted to microbial protein | Link |