Difference between revisions of "Category:Pericardial Pathology"

From WikiVet English
Jump to navigation Jump to search
(Created page with ' The heart is invested in a pericardial sac with both visceral and parietal components. The visceral layer is closely adherent to the heart wall. The space within the pericard…')
 
 
(7 intermediate revisions by 2 users not shown)
Line 1: Line 1:
−
 
+
{{frontpage
−
 
+
|pagetitle =Pericardial Pathology
 +
|pagebody = <div style="text-align: left; direction: ltr; margin-left: 1em;">
 
The heart is invested in a pericardial sac with both visceral and parietal components.  The visceral layer is closely adherent to the heart wall.  The space within the pericardium is small and contains approximately 1-5ml serous fluid.  This allows smooth movement of the heart within the pericardium during normal systole and diastole.<br>
 
The heart is invested in a pericardial sac with both visceral and parietal components.  The visceral layer is closely adherent to the heart wall.  The space within the pericardium is small and contains approximately 1-5ml serous fluid.  This allows smooth movement of the heart within the pericardium during normal systole and diastole.<br>
 
The pericardial sac is thin, fibrous and inelastic so prevents and significant distension. This causes a constrictive effect when fluid accumulates within the sac.
 
The pericardial sac is thin, fibrous and inelastic so prevents and significant distension. This causes a constrictive effect when fluid accumulates within the sac.
Line 12: Line 13:
 
*Pericardial cysts.
 
*Pericardial cysts.
 
*Partial or complete absence of the paricardial sac.
 
*Partial or complete absence of the paricardial sac.
 +
</div>
 +
|contenttitle =Content
 +
|contentbody =<big><b>
 +
<categorytree mode=pages>Pericardial Pathology</categorytree>
  
−
 
+
</b></big>
−
 
+
|logo =path-logo.png
−
==Metabolic Pathology==
+
}}
−
 
 
−
===[[Serous Atrophy of Fat - Pathology|Serous atrophy of fat]]===
 
−
 
 
−
===Epicardial mineralisation===
 
−
 
 
−
===[[Urate Metabolism - Pathology|Urate deposition]]===
 
−
 
 
−
==Non-inflammatory-Fluid accumulation==
 
−
 
 
−
Fluid accumulation within the pericardial sac may lead to fibrous thickening and opacity of the pericardium if prolonged.  Villous proliferation of the serosa will occur due to the irritation caused by the presence of the fluid.
 
−
 
 
−
Large volumes of fluid within the pericardial sac may cause [[Cardiac Tamponade - Pathology|cardiac tamponade]].
 
−
 
 
−
===Hydropericardium===
 
−
 
 
−
Serous transudate into the pericardial sac due to congestive heart failure.  Will also see ascites, hydrothorax etc also associated with right sided congestive heart failure. 
 
−
 
 
−
Hydropericardium may also be seen with:
 
−
*Neoplasia.
 
−
*Anaemia.
 
−
*General debility.
 
−
*Toxins E.g. Protein rich fluid found within the pericardium with fibrin clots seen with the clostridial enterotoxaemia [[Pulpy_Kidney|Pulpy kidney disease]].
 
−
 
 
−
===Haemopericardium===
 
−
 
 
−
Whole blood within the pericardial sac, often associated with rupture of the heart or great vessels.  Often due to rupture of the left atrium whose aetiology is unkown but postulated to be:
 
−
*Congenital.
 
−
*Idiopathic.
 
−
May also be associated with RTA trauma or ulcerative endocarditis due to uraemia in dogs with kidney failure.
 
−
 
 
−
Death usually reults due to [[Cardiac Tamponade - Pathology|cardiac tamponade]].
 
−
 
 
−
===Haemorrhagic effusion===
 
−
 
 
−
Idiopathic in giant breeds of dog E.g. St Bernard.
 
−
 
 
−
==Inflammatory-Pericarditis==
 
−
 
 
−
Usually an infective aetiology.  As with non-inflammatory accumulation of fluid the main complication is the restriction of ventricular movement.  Clinical signs seen are therefore those of circulatory failure along with pyrexia and a general depression. 
 
−
[[Image:Pericarditis-histo.jpg|right|thumb|125px|<small><center>'''Pericarditis'''. Courtesy of A. Jefferies</center></small>]]
 
−
 
 
−
Spread of infectious agent may be:
 
−
*Haematogenous; following generalised infection.  Most often seen in cattle and pigs.
 
−
*Extension of infection form surrounding tissues; for example from the lungs, pleura, mediastinum.
 
−
*Extension of infection from myocardium; rare.
 
−
*Traumatic penetration of the pericardium;
 
−
**Foreign bodies from the oesophagus or reticulum in cattle; [[Traumatic_Reticulitis|traumatic reticulo-peritonitis]].
 
−
**Fractured ribs; E.g. RTAs in small animals, horses etc.
 
−
 
 
−
Pericarditis can be subdivided into two main categories:
 
−
 
 
−
===Fibrinous pericarditis===
 
−
 
 
−
[[Image:Fibrinous pericarditis.jpg|right|thumb|125px|<small><center>'''Fibrinous pericarditis'''. Courtesy of A. Jefferies</center></small>]]
 
−
 
 
−
Most common form.  Grey strands of fibrin cover the epicardium and little fluid accumulates.  Close apposition of the parietal and visceral pericardium layers allows adhesion formation within approximatley 7-10 days.  Such adhesions may resolve with little residual pathology or may become focal or diffuse adhesive pericarditis lesions.
 
−
 
 
−
Fibrinous pericarditis produces a crackiling sound on auscultation.
 
−
 
 
−
<br><br>
 
−
 
 
−
===Suppurative pericarditis===
 
−
[[Image:traumatic pericarditis 2.jpg|right|thumb|125px|<small><center>'''Traumatic pericarditis'''. Courtesy of A. Jefferies</center></small>]]
 
−
Purulent pericarditis indicates the presence of pyogenic organisms E.g. Staphs
 
−
Usually occurs in cattle as a result of traumatic penetration of the pericardial sac with a sharp metallic object or ''wire''.  This is [[Traumatic_Reticulitis|Traumatic reticulo-peritonitis]]
 
−
Death usually occurs before organisation and a constrictive pericarditis can become apparent.
 
−
[[Image:traumatic reticulitis.jpg|right|thumb|125px|<small><center>'''Traumatic pericarditis'''. Courtesy of A. Jefferies</center></small>]]
 
−
[[Image:Traumatic pericarditis 4.jpg|right|thumb|125px|<small><center>'''Traumatic pericarditis'''. Courtesy of A. Jefferies</center></small>]]
 
−
'''Sequalae of pericarditis:'''
 
−
*Resolution with no further clinical significance.
 
−
*Adhesion: organisation of fibrin.  May lead to a ''bread and butter'' appearance.
 
−
*Constriction: gradual [[Cardiac Tamponade - Pathology|cardiac tamponade]] will occur.
 
  
 
[[Category:Cardiovascular_System_-_Pathology]]
 
[[Category:Cardiovascular_System_-_Pathology]]

Latest revision as of 18:48, 15 February 2011

Pericardial Pathology

The heart is invested in a pericardial sac with both visceral and parietal components. The visceral layer is closely adherent to the heart wall. The space within the pericardium is small and contains approximately 1-5ml serous fluid. This allows smooth movement of the heart within the pericardium during normal systole and diastole.
The pericardial sac is thin, fibrous and inelastic so prevents and significant distension. This causes a constrictive effect when fluid accumulates within the sac.

Pericardial disease accounts for only 1% of all clinically significant cardiac disease in the dog and cat, however it a frequently overlooked cause of right heart failure. Pericardial disease which is clinically significant is rare in the cat.

The most common congenital abnormality of the pericardium is Peritoneo-pericardial diaphragmatic hernia.

Other congenital abnormalities of the pericardial sac include:

  • Pericardial cysts.
  • Partial or complete absence of the paricardial sac.

Content

▼ Pericardial Pathology
  Cardiac Tamponade
  Haemopericardium
  Haemorrhagic Effusion, Pericardial
  Hernia, Peritoneopericardial Diaphragmatic
  Hydropericardium
  Pericardial Effusion
  Pericarditis
  Pericarditis, Constrictive
  Serous Atrophy of Fat - Pathology
  Pericardial Pathology Flashcards

Retrieved from "https://en.wikivet.net/index.php?title=Category:Pericardial_Pathology&oldid=107725"