Difference between revisions of "Ovaries Endocrine Function - Anatomy & Physiology"
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|<font color=Purple>Oestrogen</font> | |<font color=Purple>Oestrogen</font> | ||
− | + | |GnRH from the [[Endocrine System - Hypothalamus - Anatomy & Physiology|Hypothalamus]] causes FSH secretion from the [[Endocrine System - Pituitary Gland - Anatomy & Physiology|Pituitary Gland]] which stimulates the developing follicles in the ovary to secrete this hormone. | |
− | |GnRH from the [[Endocrine System - Hypothalamus - Anatomy & Physiology|Hypothalamus]] causes | + | |Promotes development of mammary tissue and is vital for thickening of the endometrium prior to implantation. Oestrogens regulate the [[Female Reproductive Tract -Oestrous Cycle - Anatomy & Physiology|Oestrus Cycle]]. |
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|<font color=Purple>Progesterone</font> | |<font color=Purple>Progesterone</font> | ||
− | | | + | |This is produced in the Corpus Luteum after ovulation, and also the [[Endocrine System - Placenta - Anatomy & Physiology|Placenta]] during pregnancy. |
− | + | |Progesterone is sometimes known as the ''hormone of pregnancy''. It converts the endometrium to its secretory stage to prepare for implantation, reduces the immune response within the uterus to allow for acceptance of the conceptus, and decreases contractility of the smooth muscle. | |
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|<font color=Purple>Inhibin<font> | |<font color=Purple>Inhibin<font> | ||
− | | | + | |Granulosa Cells |
− | |GnRH from the [[Endocrine System - Hypothalamus - Anatomy & Physiology|Hypothalamus]] causes FSH secretion from the [[Endocrine System - Pituitary Gland - Anatomy & Physiology|Pituitary Gland]] which stimulates the | + | |GnRH from the [[Endocrine System - Hypothalamus - Anatomy & Physiology|Hypothalamus]] causes FSH secretion from the [[Endocrine System - Pituitary Gland - Anatomy & Physiology|Pituitary Gland]] which stimulates the Granulosa cells. |
− | | | + | |Inhibits FSH secretion. |
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|<font color=Purple>Relaxin<font> | |<font color=Purple>Relaxin<font> | ||
− | | | + | |This is produced by the Corpus Luteum towards the end of pregnancy as a result of fetal ACTH production. |
− | + | |Relaxes the cervix and pelvic ligaments in preparation for parturition. | |
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|<font color=Purple>[[Endocrine System - <font> | |<font color=Purple>[[Endocrine System - <font> |
Revision as of 11:34, 3 July 2008
Reproductive hormones tend to have short half lives and act in minute quantities. They bind to specific receptors on their target organs.
Oestrus Cycle
Hormone | Produced by | Regulation | Action |
---|---|---|---|
Oestrogen | GnRH from the Hypothalamus causes FSH secretion from the Pituitary Gland which stimulates the developing follicles in the ovary to secrete this hormone. | Promotes development of mammary tissue and is vital for thickening of the endometrium prior to implantation. Oestrogens regulate the Oestrus Cycle. | |
Progesterone | This is produced in the Corpus Luteum after ovulation, and also the Placenta during pregnancy. | Progesterone is sometimes known as the hormone of pregnancy. It converts the endometrium to its secretory stage to prepare for implantation, reduces the immune response within the uterus to allow for acceptance of the conceptus, and decreases contractility of the smooth muscle. | |
Inhibin | Granulosa Cells | GnRH from the Hypothalamus causes FSH secretion from the Pituitary Gland which stimulates the Granulosa cells. | Inhibits FSH secretion. |
Relaxin | This is produced by the Corpus Luteum towards the end of pregnancy as a result of fetal ACTH production. | Relaxes the cervix and pelvic ligaments in preparation for parturition. | |
[[Endocrine System - | Sertoli Cells | GnRH from the Hypothalamus causes FSH secretion from the Pituitary Gland which stimulates the Sertoli Cells. | Converted from testosterone, this hormone's function is complex. It may prevent apoptosis of male germ cells. |