Changes

The stenotic area causing the problem is a consequence of fibrous tissue on and around the aortic valve. A narrowing of the aortic outflow tract causes an increase in pressure and this increased pressure leads to left ventricular dilation and concentric hypertrophy as a means of maintaining sufficient cardiac output. The severity of stenosis determines the extent of hypertrophy. Hypertrophy of the myocardium leads to an increased oxygen demand and decreased diastolic filling and insufficient oxygen to meet the new needs of the hypertrophied left ventricle predisposes the animal to ventricular arrhythmias. There is also an increased risk of bacterial endocarditis in animals affected by this condition.  

The stenotic area causing the problem is a consequence of fibrous tissue on and around the aortic valve. A narrowing of the aortic outflow tract causes an increase in pressure and this increased pressure leads to left ventricular dilation and concentric hypertrophy as a means of maintaining sufficient cardiac output. The severity of stenosis determines the extent of hypertrophy. Hypertrophy of the myocardium leads to an increased oxygen demand and decreased diastolic filling and insufficient oxygen to meet the new needs of the hypertrophied left ventricle predisposes the animal to ventricular arrhythmias. There is also an increased risk of bacterial endocarditis in animals affected by this condition.  

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== Clinical Signs ==

As with all heart conditions, weakness, exercise intolerance, syncope, stunting of growth and pallor are all possible clinical signs. However, the disease may be asymptomatic.<br>  

As with all heart conditions, weakness, exercise intolerance, syncope, stunting of growth and pallor are all possible clinical signs. However, the disease may be asymptomatic.<br>