Author, Donkey
201
edits
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|38
|38
|(32 - 48)
|(32 - 48)
|'''Globulins.''' Acute phase proteins. Inflammatory processes usually result in increases in globulins. Serum protein electrophoresis is rarely indicated except in the investigation of certain neoplastic processes. Hypoglobulinaemia may be seen subsequent to infectious disease processes but is rarely of relevance. Hyperglobulinaemia may also be seen in cases of hepatopathy.
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!align="left" bgcolor="#F2F2F2"|Creat μmol/l
!align="left" bgcolor="#F2F2F2"|Creat μmol/l
|bgcolor="#F2F2F2"|87
|bgcolor="#F2F2F2"|87
|bgcolor="#F2F2F2"|(53 - 118)
|bgcolor="#F2F2F2"|(53 - 118)
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|bgcolor="#F2F2F2"| '''Creatinine.''' Produced in the body by muscle breakdown and filtered by the kidneys. A marker of renal function. In equines creatinine is the most reliable marker of glomerular filtration rate as tubular reabsorption is minimal. Renal tubular damage and reduced glomerular filtration rates will result in increased creatinine.
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!align="left"|Urea mmol/l
!align="left"|Urea mmol/l
|3.2
|3.2
|(1.5 – 5.2)
|(1.5 – 5.2)
|'''Urea.''' Relevant to renal and hepatic function. Traditionally used as a marker of glomerular filtration rate but is subject to enterohepatic recycling and has variable reabsorption in the renal tubules. Elevations in creatinine due to renal dysfunction are more reliable than changes in urea.
Urea is produced in the liver. Reduced hepatic function may result in decreases in urea.
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!align="left" bgcolor="#F2F2F2"|Amylase iu/l
!align="left" bgcolor="#F2F2F2"|Amylase iu/l