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Lungs Inflammatory - Pathology (view source)
Revision as of 17:38, 23 February 2010
, 17:38, 23 February 2010no edit summary
*Can result in [[Bronchi and Bronchioles Inflammatory - Pathology#Diffuse Fibrosing Alveolitis/ Interstitial Pneumonia|diffuse fibrosing alveolitis]]
*Can result in [[Bronchi and Bronchioles Inflammatory - Pathology#Diffuse Fibrosing Alveolitis/ Interstitial Pneumonia|diffuse fibrosing alveolitis]]
*Gross appearance is that of enlarged wet lungs, the interlobular septa are markedly widenedith [[Lungs Circulatory - Pathology#Pulmonary oedema|oedema]] and [[Lungs Ventilation - Pathology#Emphysema|emphysema]]
*Gross appearance is that of enlarged wet lungs, the interlobular septa are markedly widenedith [[Lungs Circulatory - Pathology#Pulmonary oedema|oedema]] and [[Lungs Ventilation - Pathology#Emphysema|emphysema]]
*Underlying pathogenesis is ingestion of L-tryptophan in the pasture which is metabolised to 3-methylindole -> bloodstream -> lungs -> metabolised into a compound toxic to Type 1 pneu[[Monocytes - WikiBlood]] and non-ciliated bronchiolar epithelium
*Underlying pathogenesis is ingestion of L-tryptophan in the pasture which is metabolised to 3-methylindole -> bloodstream -> lungs -> metabolised into a compound toxic to Type 1 pneumonocytes and non-ciliated bronchiolar epithelium
*Their loss allows massive flooding of the alveoli with a protein-rich fluid
*Their loss allows massive flooding of the alveoli with a protein-rich fluid
*The sequence of events in the lung is as follows:
*The sequence of events in the lung is as follows: