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|linktext = WikiQuiz
|linktext = WikiQuiz
|pagetype=Quiz
|pagetype=Quiz
|Review= '''Michael Jones''' BSc (Hons) MSc PhD Lecturer in Microbiology and Molecular Biology
|Review= '''Michael Jones''' BSc (Hons) MSc PhD Lecturer in Microbiology and Molecular Biology}}
}}
{{QuizCat|topic=Bugs|topicsubcategory=B}}
{{QuizCat|topic=Bugs|topicsubcategory=B}}
{{QuizCat|topic=Pathology|topicsubcategory=B}}
{{QuizCat|topic=Pathology|topicsubcategory=B}}
feedback5="'''Incorrect.''' Lawsonia infection can only take place in the presence of intestinal flora. There is a synergistic relationship between L. intracellularis and intestinal flora including E. coli, Clostridium species and Bacteroides species.[[ Lawsonia intracellularis|WikiVet Article: Lawsonia ]]"
feedback5="'''Incorrect.''' Lawsonia infection can only take place in the presence of intestinal flora. There is a synergistic relationship between L. intracellularis and intestinal flora including E. coli, Clostridium species and Bacteroides species.[[ Lawsonia intracellularis|WikiVet Article: Lawsonia ]]"
feedback3="'''Incorrect.''' Lawsonia infection can only take place in the presence of intestinal flora. There is a synergistic relationship between L. intracellularis and intestinal flora including E. coli, Clostridium species and Bacteroides species.[[Lawsonia intracellularis |WikiVet Article: Lawsonia]]"
feedback3="'''Incorrect.''' Lawsonia infection can only take place in the presence of intestinal flora. There is a synergistic relationship between L. intracellularis and intestinal flora including E. coli, Clostridium species and Bacteroides species.[[Lawsonia intracellularis |WikiVet Article: Lawsonia]]"
feedback1="'''Incorrect.''' Lawsonia infection can only take place in the presence of intestinal flora. There is a synergistic relationship between L. intracellularis and intestinal flora including E. coli, Clostridium species and Bacteroides species.Somatostatin is growth hormone inhibiting hormone which is secreted in several locations in the gastrointestinal tract. [[Lawsonia intracellularis http:/ / en.wikipedia.org/ wiki/ Somatostatin |WikiVet Article: LawsoniaLink to Wikipedia: Somatostatin ]]"
feedback1="'''Incorrect.''' Lawsonia infection can only take place in the presence of intestinal flora. There is a synergistic relationship between L. intracellularis and intestinal flora including E. coli, Clostridium species and Bacteroides species.Somatostatin is growth hormone inhibiting hormone which is secreted in several locations in the gastrointestinal tract. [[Lawsonia intracellularis|WikiVet Article: Lawsonia]]"
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choice2="Goblet cells"
choice2="Goblet cells"
correctchoice="3"
correctchoice="3"
feedback3="'''Correct!''' Lawsonia intracellularis has an affinity for enterocytes. Enterocytes are simple columnar epithelium cells of the small intestine and colon. They are thought to have a a secretory role. [[Lawsonia intracellularis http:/ / en.wikipedia.org/ wiki/ Enterocyte|WikiVet Article: Lawsonia intracellularis. Link to Wikipedia: Enterocyte]]"
feedback3="'''Correct!''' Lawsonia intracellularis has an affinity for enterocytes. Enterocytes are simple columnar epithelium cells of the small intestine and colon. They are thought to have a a secretory role. [[Lawsonia intracellularis |WikiVet Article: Lawsonia intracellularis]]"
feedback4="'''Incorrect.''' Lawsonia intracellularis is not known to have an affinity for paneth cells. Paneth cells are also found in the intestinal tract and contain zinc and lysoyme. The correct answer is that Lawsonia intracellularis has an affinity for enterocytes. Enterocytes are simple columnar epithelium cells of the small intestine and colon. They are thought to have a a secretory role. [[Lawsonia intracellularis http:/ / en.wikipedia.org/ wiki/ Enterocytehttp:/ / en.wikipedia.org/ wiki/ Paneth cell|WikiVet Article: Lawsonia intracellularis. Link to Wikipedia: Enterocyte, Paneth cell.]]"
feedback4="'''Incorrect.''' Lawsonia intracellularis is not known to have an affinity for paneth cells. Paneth cells are also found in the intestinal tract and contain zinc and lysoyme. The correct answer is that Lawsonia intracellularis has an affinity for enterocytes. Enterocytes are simple columnar epithelium cells of the small intestine and colon. They are thought to have a a secretory role. [[Lawsonia intracellularis|WikiVet Article: Lawsonia intracellularis]]"
feedback5="'''Incorrect.''' Lawsonia intracellularis is not known to have an affinity for parietal cells. Parietal are part of the stomach epithelium and secrete gastric acid and intrinsic factor. The correct answer is that Lawsonia intracellularis has an affinity for enterocytes. Enterocytes are simple columnar epithelium cells of the small intestine and colon. They are thought to have a a secretory role. [[Lawsonia intracellularis http:/ / en.wikipedia.org/ wiki/ Enterocytehttp:/ / en.wikipedia.org/ wiki/ Parietal cell|WikiVet Article: Lawsonia intracellularis. Link to Wikipedia: Enterocyte, Parietal cell.]]"
feedback5="'''Incorrect.''' Lawsonia intracellularis is not known to have an affinity for parietal cells. Parietal are part of the stomach epithelium and secrete gastric acid and intrinsic factor. The correct answer is that Lawsonia intracellularis has an affinity for enterocytes. Enterocytes are simple columnar epithelium cells of the small intestine and colon. They are thought to have a a secretory role. [[Lawsonia intracellularis |WikiVet Article: Lawsonia intracellularis]]"
feedback1="'''Incorrect.''' Lawsonia intracellularis is not known to have an affinity for enteroendocrine cells. Enteroendocrine cells are specialized endocrine cell of the GI tract. The correct answer is that Lawsonia intracellularis has an affinity for enterocytes. Enterocytes are simple columnar epithelium cells of the small intestine and colon. They are thought to have a a secretory role. [[Lawsonia intracellularis http:/ / en.wikipedia.org/ wiki/ Enterocyte http:/ / en.wikipedia.org/ wiki/ Enteroendocrine cell |WikiVet Article: Lawsonia intracellularis. Link to Wikipedia: Enterocyte, Enteroendocrine cell.]]"
feedback1="'''Incorrect.''' Lawsonia intracellularis is not known to have an affinity for enteroendocrine cells. Enteroendocrine cells are specialized endocrine cell of the GI tract. The correct answer is that Lawsonia intracellularis has an affinity for enterocytes. Enterocytes are simple columnar epithelium cells of the small intestine and colon. They are thought to have a a secretory role. [[Lawsonia intracellularis |WikiVet Article: Lawsonia intracellularis]]"
feedback2="'''Incorrect.''' Lawsonia intracellularis is not known to have an affinity for goblet cells. Goblet cells are glandular simple columnar epithelial cells that secrete mucus. The correct answer is that Lawsonia intracellularis has an affinity for enterocytes. Enterocytes are simple columnar epithelium cells of the small intestine and colon. They are thought to have a a secretory role. [[Lawsonia intracellularis http:/ / en.wikipedia.org/ wiki/ Enterocytehttp:/ / en.wikipedia.org/ wiki/ Goblet cell|WikiVet Article: Lawsonia intracellularis. Link to Wikipedia: Enterocyte, Goblet cell]]"
feedback2="'''Incorrect.''' Lawsonia intracellularis is not known to have an affinity for goblet cells. Goblet cells are glandular simple columnar epithelial cells that secrete mucus. The correct answer is that Lawsonia intracellularis has an affinity for enterocytes. Enterocytes are simple columnar epithelium cells of the small intestine and colon. They are thought to have a a secretory role. [[Lawsonia intracellularis|WikiVet Article: Lawsonia intracellularis]]"
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choice4="Proteases"
choice4="Proteases"
correctchoice="3"
correctchoice="3"
feedback3="'''Correct!''' Elastase is produced by Pseudomonas aeruginosa and damages elastin in lung parenchyma and blood vessel walls. This aids in the disease process which in many circumstances leads to pneumonia. [[ Pseudomonas aeruginosa and Burkholderia species|WikiVet Article: Pseudomonas]]"
feedback3="'''Correct!''' Elastase is produced by Pseudomonas aeruginosa and damages elastin in lung parenchyma and blood vessel walls. This aids in the disease process which in many circumstances leads to pneumonia. [[:Category:Pseudomonas and Burkholderia species|WikiVet Article: Pseudomonas]]"
feedback2="'''Incorrect.''' LPS is a component of the outer membrane of bacteria and is not released by bacteria until death. Pseudomonas resists phagocytosis and complement via its LPS. The correct answer is elastase which is produced by Pseudomonas aeruginosa and damages elastin in lung parenchyma and blood vessel walls. This aids in the disease process which in many circumstances leads to pneumonia. [[ Pseudomonas aeruginosa and Burkholderia species|WikiVet Article: Pseudomonas]]"
feedback2="'''Incorrect.''' LPS is a component of the outer membrane of bacteria and is not released by bacteria until death. Pseudomonas resists phagocytosis and complement via its LPS. The correct answer is elastase which is produced by Pseudomonas aeruginosa and damages elastin in lung parenchyma and blood vessel walls. This aids in the disease process which in many circumstances leads to pneumonia. [[:Category:Pseudomonas and Burkholderia species|WikiVet Article: Pseudomonas]]"
feedback1="'''Incorrect.''' Exotoxin A, phospholipase C and proteases, produced by Pseudomonas, allow tissue invasion and damage, but not of the lung parenchyma.The correct answer is elastase which is produced by Pseudomonas aeruginosa and damages elastin in lung parenchyma and blood vessel walls. This aids in the disease process which in many circumstances leads to pneumonia. [[ Pseudomonas aeruginosa and Burkholderia species|WikiVet Article: Pseudomonas ]]"
feedback1="'''Incorrect.''' Exotoxin A, phospholipase C and proteases, produced by Pseudomonas, allow tissue invasion and damage, but not of the lung parenchyma.The correct answer is elastase which is produced by Pseudomonas aeruginosa and damages elastin in lung parenchyma and blood vessel walls. This aids in the disease process which in many circumstances leads to pneumonia. [[:Category:Pseudomonas and Burkholderia species|WikiVet Article: Pseudomonas ]]"
feedback5="'''Incorrect.''' Exotoxin A, phospholipase C and proteases, produced by Pseudomonas, allow tissue invasion and damage, but not of the lung parenchyma. The correct answer is elastase which is produced by Pseudomonas aeruginosa and damages elastin in lung parenchyma and blood vessel walls. This aids in the disease process which in many circumstances leads to pneumonia. [[Pseudomonas aeruginosa and Burkholderia species |WikiVet Article: Pseudomonas ]]"
feedback5="'''Incorrect.''' Exotoxin A, phospholipase C and proteases, produced by Pseudomonas, allow tissue invasion and damage, but not of the lung parenchyma. The correct answer is elastase which is produced by Pseudomonas aeruginosa and damages elastin in lung parenchyma and blood vessel walls. This aids in the disease process which in many circumstances leads to pneumonia. [[:Category:Pseudomonas and Burkholderia species|WikiVet Article: Pseudomonas ]]"
feedback4="'''Incorrect.''' Exotoxin A, phospholipase C and proteases, produced by Pseudomonas, allow tissue invasion and damage, but not of the lung parenchyma. The correct answer is elastase which is produced by Pseudomonas aeruginosa and damages elastin in lung parenchyma and blood vessel walls. This aids in the disease process which in many circumstances leads to pneumonia. [[Pseudomonas aeruginosa and Burkholderia species |WikiVet Article: Pseudomonas ]]"
feedback4="'''Incorrect.''' Exotoxin A, phospholipase C and proteases, produced by Pseudomonas, allow tissue invasion and damage, but not of the lung parenchyma. The correct answer is elastase which is produced by Pseudomonas aeruginosa and damages elastin in lung parenchyma and blood vessel walls. This aids in the disease process which in many circumstances leads to pneumonia. [[:Category:Pseudomonas and Burkholderia species|WikiVet Article: Pseudomonas ]]"
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</WikiQuiz>
choice2="Secondary active transport"
choice2="Secondary active transport"
correctchoice="1"
correctchoice="1"
feedback1="'''Correct!'''Enteroinvasive E. coli induce receptor-mediated endocytosis in the epithelial cells of the intestine. [[Escherichia colihttp:/ / en.wikipedia.org/ wiki/ Endocytosis|WikiVet Article: E. coliLink to Wikipedia: Endocytosis]]"
feedback1="'''Correct!''' Enteroinvasive E. coli induce receptor-mediated endocytosis in the epithelial cells of the intestine. [[Escherichia coli|WikiVet Article: E. coli]]"
feedback4="'''Incorrect.''' Pinocytosis is concerned with the uptake of solutes and single molecules such as proteins. Enteroinvasive E. coli induce receptor-mediated endocytosis in the epithelial cells of the intestine. [[Escherichia coli http:/ / en.wikipedia.org/ wiki/ Pinocytosis |WikiVet Article: E. coli. Link to Wikipedia: Pinocytosis.]]"
feedback4="'''Incorrect.''' Pinocytosis is concerned with the uptake of solutes and single molecules such as proteins. Enteroinvasive E. coli induce receptor-mediated endocytosis in the epithelial cells of the intestine. [[Escherichia coli|WikiVet Article: E. coli]]"
feedback3="'''Incorrect.''' Facilitated diffusion is a form of passive transport facilitated by transport proteins. Enteroinvasive E. coli induce receptor-mediated endocytosis in the epithelial cells of the intestine. [[Escherichia coli http:/ / en.wikipedia.org/ wiki/ Facilitated diffusion |WikiVet Article: E. coliLink to Wikipedia: Facilitated diffusion ]]"
feedback3="'''Incorrect.''' Facilitated diffusion is a form of passive transport facilitated by transport proteins. Enteroinvasive E. coli induce receptor-mediated endocytosis in the epithelial cells of the intestine. [[Escherichia coli|WikiVet Article: E. coli]]"
feedback5="'''Incorrect.''' Primary active transport uses energy (ATP) to transport molecules across a membrane. Enteroinvasive E. coli induce receptor-mediated endocytosis in the epithelial cells of the intestine. [[ Escherichia coli http:/ / en.wikipedia.org/ wiki/ Primary active transport|WikiVet Article: E. coliLink to Wikipedia: Primary active transport]]"
feedback5="'''Incorrect.''' Primary active transport uses energy (ATP) to transport molecules across a membrane. Enteroinvasive E. coli induce receptor-mediated endocytosis in the epithelial cells of the intestine. [[Escherichia coli|WikiVet Article: E. coli]]"
feedback2="'''Incorrect.''' In secondary active transport the electrochemical potential difference created by pumping ions out of the cell is used for movement. Enteroinvasive E. coli induce receptor-mediated endocytosis in the epithelial cells of the intestine. [[Escherichia colihttp:/ / en.wikipedia.org/ wiki/ Secondary active transport|WikiVet Article: E. coliLink to Wikipedia: Secondary active transport ]]"
feedback2="'''Incorrect.''' In secondary active transport the electrochemical potential difference created by pumping ions out of the cell is used for movement. Enteroinvasive E. coli induce receptor-mediated endocytosis in the epithelial cells of the intestine. [[Escherichia coli|WikiVet Article: E. coli]]"
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choice5="Type B causes the most infections in domestic animals"
choice5="Type B causes the most infections in domestic animals"
correctchoice="1"
correctchoice="1"
feedback1="'''Correct!''' Francisella tularensis causes a lymphadenitis and septicaemia in wildlife, domestic animals and humans, known as Tularaemia. Type A is highly virulent where as type B is less virulent."
feedback1="'''Correct!''' Francisella tularensis causes a lymphadenitis and septicaemia in wildlife, domestic animals and humans, known as Tularaemia. Type A is highly virulent where as type B is less virulent. [[Francisella tularensis|WikiVet Article: Francisella tularensis]]"
feedback2="'''Incorrect.''' Francisella tularensis causes a lymphadenitis and septicaemia in wildlife, domestic animals and humans, known as Tularaemia. Type A is a terrestrial strain with reservoirs in lagomorphs, rodents, galliforms and birds.Type B is waterborne with reservoirs in water, mud and affects aquatic animals. The correct answer is that type A is highly virulent and type B is less virulent."
feedback2="'''Incorrect.''' Francisella tularensis causes a lymphadenitis and septicaemia in wildlife, domestic animals and humans, known as Tularaemia. Type A is a terrestrial strain with reservoirs in lagomorphs, rodents, galliforms and birds.Type B is waterborne with reservoirs in water, mud and affects aquatic animals. The correct answer is that type A is highly virulent and type B is less virulent. [[Francisella tularensis|WikiVet Article: Francisella tularensis]]" "
feedback3="'''Incorrect.''' Francisella tularensis causes a lymphadenitis and septicaemia in wildlife, domestic animals and humans, known as Tularaemia. Type A is confined to North America, whereas type B is found in both Eurasia AND North America. The correct answer is that type A is highly virulent and type B is less virulent."
feedback3="'''Incorrect.''' Francisella tularensis causes a lymphadenitis and septicaemia in wildlife, domestic animals and humans, known as Tularaemia. Type A is confined to North America, whereas type B is found in both Eurasia AND North America. The correct answer is that type A is highly virulent and type B is less virulent. [[Francisella tularensis|WikiVet Article: Francisella tularensis]]" "
feedback4="'''Incorrect.''' Francisella tularensis causes a lymphadenitis and septicaemia in wildlife, domestic animals and humans, known as Tularaemia.Type A causes severe disease and type B causes mild disease. The correct answer is type A is highly virulent and type B is less virulent."
feedback4="'''Incorrect.''' Francisella tularensis causes a lymphadenitis and septicaemia in wildlife, domestic animals and humans, known as Tularaemia.Type A causes severe disease and type B causes mild disease. The correct answer is type A is highly virulent and type B is less virulent. [[Francisella tularensis|WikiVet Article: Francisella tularensis]]" "
feedback5="'''Incorrect.''' Francisella tularensis causes a lymphadenitis and septicaemia in wildlife, domestic animals and humans, known as Tularaemia. Type A causes the most infections in domestic animals. The correct answer is type A is highly virulent and type B is less virulent."
feedback5="'''Incorrect.''' Francisella tularensis causes a lymphadenitis and septicaemia in wildlife, domestic animals and humans, known as Tularaemia. Type A causes the most infections in domestic animals. The correct answer is type A is highly virulent and type B is less virulent. [[Francisella tularensis|WikiVet Article: Francisella tularensis]]" "
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