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| − | ===''[[Clostridium perfringens type C]]===
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| − | ===''C. perfringens'' type C===
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| − | ===''C. perfringens'' type D=== | |
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| − | *[[Intestines Catarrhal Enteritis - Pathology#"Pulpy Kidney" Disease|Pulpy kidney disease]] in well-fed 3-10 week-old lambs
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| − | *Follows overeating high grain diet or luchious pasture
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| − | *Starch from partially digested food enterering the intestine from the rumen allows rapid clostridial proliferation
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| − | *Epsilon toxin activated by proteolytic enzymes causes toxaemia
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| − | *Epsilon toxin increases intestinal and capillary permeability; also alpha toxin
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| − | *Lambs found dead or with opisthotonos, convulsions, coma in acute phases
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| − | *Blindness and head pressing in subacute disease; bloat in later stages
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| − | *Hyperglycaemia, glycosuria
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| − | *Post mortem: hyperaemia in intestine; fluid in pericardial sac; kidney autolysis with pulpy cortical softening (acute death)
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| − | *Subacute death causes symmetrical encephalomalacia and haemorrhage in basal ganglia and midbrain
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| − | *Enterotoxaemia in kids and adult goats
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| − | ===''C. perfringens'' type E===
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| − | *Enteritis in rabbits, haemorrhagic enteritis in calves
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| − | *ALpha and iota toxins
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| | ===Treatment and control of enterotoxaemic infections=== | | ===Treatment and control of enterotoxaemic infections=== |
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| | *Vaccination of lambs with toxoid before 2 months of age to protect against pulpy kidney | | *Vaccination of lambs with toxoid before 2 months of age to protect against pulpy kidney |
| | *Avoid sudden dietary changes | | *Avoid sudden dietary changes |
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| | [[Category:Clostridium_species]] | | [[Category:Clostridium_species]] |
| | + | [[Category:To_Do_-_Bacteria]] |