The most significant disease vectors for each serotype are:
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Vectors transmit viral particles between sylvatic hosts when takin ga blood meal. If the virus can peentrate the gut of the vector, then it may pass throhg the haemolymph to the oral glands, multiply and subsequntrly be shed in the saliva and other oral secretions. If th eblood meal octnains adequate numbers of viral particles, multiplication may not be rrequired for transmission. It is likely tha the mosquito remians infected for life. The most significant disease vectors for each serotype are:
''Culiseta melanura'' is another vector for Eastern EEV. It feeds mostly on swamp birds, completing an enzootic cycle of viral transmission. ''C.melanura'' is thus an inhabitant of freshwater swamps and is not usually found in areas densely populated by equids. ''Aedes'' spp. are more important in epizootics and epidemics. Culex tarsalis is the primary vector that maintains WEE virus in an enzootic cycle with passerine birds. Dermacentor andersoni ticks, Triatoma sanguisuga (assassin bug), and the cliff swallow bug (Oeciacus vicarius) may also be involved as vectors or overwintering reservoirs fro WEE. Several species of mosquitoes from at least 11 geerna have been determined ot be naturally infected with epidemic strains of VEE virus. Ticks may also be capable of viral trasnmission.
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''Culiseta melanura'' is another vector for Eastern EEV. It feeds mostly on swamp birds, completing an enzootic cycle of viral transmission. ''C.melanura'' is thus an inhabitant of freshwater swamps and is not usually found in areas densely populated by equids. Epizootics and epidemics of Eastern EEV disease are propagated by ''Aedes'' spp. Western EEV persists in an enzootic cycle with passerine birds, transmitted by ''C.tarsalis''. Other vectors or overwintering hosts for this serotype may include ''Dermacentor andersoni'' ticks, ''Triatoma sanguisuga'' (the assassin bug), and the cliff swallow bug (''Oeciacus vicarius''). Several species of mosquitoes from at least 11 geerna have been determined ot be naturally infected with epidemic strains of VEE virus. Ticks may also be capable of viral trasnmission.
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Vectors transmit viral particles between sylvatic hosts when takin ga blood meal. If the virus can peentrate the gut of the vector, then it may pass throhg the haemolymph to the oral glands, multiply and subsequntrly be shed in the saliva and other oral secretions. If th eblood meal octnains adequate numbers of viral particles, multiplication may not be rrequired for transmission. It is likely tha the mosquito remians infected for life.