Difference between revisions of "Glossitis, Ulcerative"
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m (Text replace - "Oral Cavity - Tongue - Anatomy & Physiology" to "Tongue - Anatomy & Physiology") |
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*Calici virus infection may be followed by autoimmune / exaggerated immune response which produces lesions. | *Calici virus infection may be followed by autoimmune / exaggerated immune response which produces lesions. | ||
*Will heal eventually, but takes time and cat loses lots of condition. | *Will heal eventually, but takes time and cat loses lots of condition. | ||
| − | [[Category:Tongue_-_Pathology]][[Category:Cat]] | + | [[Category:Tongue_-_Pathology]][[Category:Oral Diseases - Cat]] |
[[Category:To_Do_-_Clinical]] | [[Category:To_Do_-_Clinical]] | ||
Revision as of 15:00, 20 December 2010
- Cat
- Relatively common.
- Youngish to middle age (mainly - although can be any age).
- Unable to eat although wants to. Hurts to open mouth.
- Whole of back of mouth covered in ulcerative granulation tissue.
- Chronic, tends to start at angle of jaw (bilateral), progression quite slow but over weeks affects whole of back of mouth.
- May also affect anatomy and physiology of the tongue.
Pathology
Epithelium lost over whole of mucosa, lots of macrophages, lymphocytes and proliferating fibrous response at base.
Pathogenesis
- Probably viral.
- Calicivirus found in 7-80%, also often find picorna virus and Feline Immunodeficiency Virus
- Probably calici virus that starts it.
- Also owners often claim develops after 'having cold' prior to it.
- Calici virus infection may be followed by autoimmune / exaggerated immune response which produces lesions.
- Will heal eventually, but takes time and cat loses lots of condition.