Difference between revisions of "Adrenogenital Syndrome"

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m (Text replace - "-_Anatomy_%26_Physiology" to "- Anatomy & Physiology")
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|backcolour =EED2EE
 
|linkpage =Reproductive System - Anatomy & Physiology
 
|linktext =Reproductive System
 
|maplink = Reproductive System (Content Map) - Anatomy & Physiology
 
|sublink1=Reproductive System - Anatomy & Physiology#Fertilisation.2C Implantation and Early Embryonic Development
 
|subtext1=FERTILISATION , IMPLANTATION AND EARLY EMBRYONIC DEVELOPMENT
 
|sublink2=Fertilisation, Implantation and Early Embryonic Development -Sexual Differentiation- Anatomy & Physiology
 
|subtext2=SEXUAL DIFFERENTIATION
 
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== Aetiology ==
 
== Aetiology ==
  
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[[Category:Reproductive System]]
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[[Category:Reproductive Disorders]]

Revision as of 14:27, 7 December 2010

Aetiology

  • Genotype is XX as should be female
  • Ovaries develop normally
  • Foetal Adrenal Gland is hyperactive and secretes large quantities of Adrenal Androgens, due to a 21 hydroxylase mutation.


Consequence

  • Wolffian Ducts persist due to the presence of Androgens, thus allowing development of male internal and external genitalia.
  • Mullarian Ducts persist due to the absence of testes, hence absence of Sertoli cell MIH secretion. This allows formation of female internal genitalia.


Morphology

  • Appears male with a micropenis and scrotum
  • Both male and female internal genitalia