Difference between revisions of "Laryngeal Paralysis"
Jump to navigation
Jump to search
(Created page with "*Axonal degeneration and subsequent dysfunction of the recurrent laryngeal nerve -> atrophy and functional paralysis of the cricoarytenoid muscle which is the main abductor of th...") |
|||
| Line 6: | Line 6: | ||
**Usually associated with other clinically apparent neurological or electromyographic defects | **Usually associated with other clinically apparent neurological or electromyographic defects | ||
**May be caused by: | **May be caused by: | ||
| − | ***[[ | + | ***[[Hypothyroidism|Hypothyroidism]] |
***Toxins - organophosphate toxicity, lead poisoning | ***Toxins - organophosphate toxicity, lead poisoning | ||
***Congenital abnormalities of the recurrent laryngeal nerve and brain stem - young age of onset | ***Congenital abnormalities of the recurrent laryngeal nerve and brain stem - young age of onset | ||
Revision as of 14:38, 21 February 2011
- Axonal degeneration and subsequent dysfunction of the recurrent laryngeal nerve -> atrophy and functional paralysis of the cricoarytenoid muscle which is the main abductor of the arytenoid cartilage -> inability to abduct arytenoid cartilage -> incomplete dilation of the larynx during inspiration and the flaccid arytenoid cartilage can be sucked into the laryngeal lumen during inspiration -> inspiratory dyspnoea
- Caused by idiopathic neuronal degeneration
- In Dogs
- Mainly large breeds eg: Labradors
- Usually bilateral disease
- Usually associated with other clinically apparent neurological or electromyographic defects
- May be caused by:
- Hypothyroidism
- Toxins - organophosphate toxicity, lead poisoning
- Congenital abnormalities of the recurrent laryngeal nerve and brain stem - young age of onset
- Secondary compression or inflammation of the recurrent laryngeal nerve