Difference between revisions of "Trichostrongylus axei"
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The life cycle of ''T. axei'' is typical of a trichostrongyloid, and is direct. The eggs develop into infective L3 in around one week. The larvae are then ingested by the host, and the larvae become exsheathed. They penetrate the mucosa of the small intestine. There are two further moults, and then the larve become adults. | The life cycle of ''T. axei'' is typical of a trichostrongyloid, and is direct. The eggs develop into infective L3 in around one week. The larvae are then ingested by the host, and the larvae become exsheathed. They penetrate the mucosa of the small intestine. There are two further moults, and then the larve become adults. | ||
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==== Pathogenicity ==== | ==== Pathogenicity ==== | ||
| − | + | Similar to ''Ostertagia'', except L3 penetrate mucosa between gastric glands. Leads to hyperplastic nodular lesions (several cms diameter) and erosions where epithelium is lost. Not usually a primary pathogen in horses. | |
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[[Category:Trichostrongyloidea]][[Category:Horse_Nematodes]] | [[Category:Trichostrongyloidea]][[Category:Horse_Nematodes]] | ||
[[Category:To_Do_-_Max]] | [[Category:To_Do_-_Max]] | ||
Revision as of 12:00, 17 July 2010
| Also known as: | Trichostrongylus extenuatus |
Hosts
Cattle, sheep, deer, horse, goat, donkey, pig and occasionally man.
Identification
T. axei are small trichostrongyle, typically aroun 5mm. They are grey when young, but become reddish brown when they mature. The spiculus are dissimilar and are not all of the same length.
Life-Cycle
The life cycle of T. axei is typical of a trichostrongyloid, and is direct. The eggs develop into infective L3 in around one week. The larvae are then ingested by the host, and the larvae become exsheathed. They penetrate the mucosa of the small intestine. There are two further moults, and then the larve become adults.
Pathogenicity
Similar to Ostertagia, except L3 penetrate mucosa between gastric glands. Leads to hyperplastic nodular lesions (several cms diameter) and erosions where epithelium is lost. Not usually a primary pathogen in horses.