Difference between revisions of "Gastritis, Catarrhal"
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m (Text replace - "[[Forestomach - Anatomy & Physiology|" to "[[Monogastric Stomach - Anatomy & Physiology|") |
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** Infiltration of inflammatory cells | ** Infiltration of inflammatory cells | ||
** No fibrin or haemorrhage. | ** No fibrin or haemorrhage. | ||
− | * The surface of the mucosa is covered by a white, sticky catarrhal exudate which lines the [[ | + | * The surface of the mucosa is covered by a white, sticky catarrhal exudate which lines the [[Monogastric Stomach - Anatomy & Physiology|stomach]]. |
===Pathogenesis=== | ===Pathogenesis=== |
Revision as of 13:15, 6 September 2010
Clinical
- Catarrhal gastritis can be fatal since it makes the animal vomit and can produce rapid dehydration.
- May die in day or two if vomiting is persistent and untreated.
- Extracellular fluid (isontonic) is lost, and so blood very quickly becomes viscous.
- Death may occur from hypovolaemic shock
- Particularly in young animals (can be very quick).
- Death may occur from hypovolaemic shock
Pathology
- The mucosa appears swollen and hyperaemic, with thickened rugae.
- Mild inflammation, hyperaemia, and oedema
- Infiltration of inflammatory cells
- No fibrin or haemorrhage.
- The surface of the mucosa is covered by a white, sticky catarrhal exudate which lines the stomach.
Pathogenesis
- There are numerous causes of catarrhal gastritis
- Ingestion of mild irritant
- Systemic bacterial diseases
- Infectious enteric diseases e.g.
- Transmissible gastro enteritis (TGE)
- E.coli
- Salmonella etc.
- Dogs are very prone catarrhal gastritis.