Difference between revisions of "Gastritis, Catarrhal"

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m (Text replace - "[[Forestomach - Anatomy & Physiology|" to "[[Monogastric Stomach - Anatomy & Physiology|")
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** Infiltration of inflammatory cells  
 
** Infiltration of inflammatory cells  
 
** No fibrin or haemorrhage.  
 
** No fibrin or haemorrhage.  
* The surface of the mucosa is covered by a white, sticky catarrhal exudate which lines the [[Forestomach - Anatomy & Physiology|stomach]].
+
* The surface of the mucosa is covered by a white, sticky catarrhal exudate which lines the [[Monogastric Stomach - Anatomy & Physiology|stomach]].
  
 
===Pathogenesis===
 
===Pathogenesis===

Revision as of 13:15, 6 September 2010

Clinical

  • Catarrhal gastritis can be fatal since it makes the animal vomit and can produce rapid dehydration.
    • May die in day or two if vomiting is persistent and untreated.
  • Extracellular fluid (isontonic) is lost, and so blood very quickly becomes viscous.
    • Death may occur from hypovolaemic shock
      • Particularly in young animals (can be very quick).

Pathology

  • The mucosa appears swollen and hyperaemic, with thickened rugae.
    • Mild inflammation, hyperaemia, and oedema
    • Infiltration of inflammatory cells
    • No fibrin or haemorrhage.
  • The surface of the mucosa is covered by a white, sticky catarrhal exudate which lines the stomach.

Pathogenesis

  • There are numerous causes of catarrhal gastritis
    1. Ingestion of mild irritant
    2. Systemic bacterial diseases
    3. Infectious enteric diseases e.g.
      • Transmissible gastro enteritis (TGE)
      • E.coli
      • Salmonella etc.
  • Dogs are very prone catarrhal gastritis.
    • Anything that affects dogs tends to produce vomiting.
    • Dogs eat almost anything and this can make them vomit for a short time.