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Pulmonary Congenital Atelectasis (view source)
Revision as of 16:54, 19 February 2011
, 16:54, 19 February 2011Created page with "*Incomplete expansion of the lung at birth (congenital) or collapse of previously air-filled lung (acquired) *Failure of ..."
*Incomplete expansion of the lung at birth (congenital) or collapse of previously air-filled lung ([[Lungs Ventilation - Pathology#Atelectasis (Collapse)|acquired]])
*Failure of lung tissue to expand at birth
*Ranges from total in stillborn animals to patchy areas in the case of weak neonates, showing more in the dependant lobes in recumbency
*'''The neonatal respiratory syndrome'''
**Best documented in foals but reported in other species
**Also features extensive atelectasis
**These animals are called 'barkers' due to the noise made during forced expiration
*Gross pathology:
**Red fleshy appearance to the affected part, depressed below the surface if there are adjacent normal aerated areas
**Cut surface oozes blood, perhaps some froth in 'barkers'
**Affected areas sink or are submerged in fixative
*Micro pathology:
**Close approximation of capillary walls
**Little normal alveolar pattern
**'Barkers' may have a proteinaceous fluid in alveoli with hyaline membranes in lower airways
*Cause:
**In weak neonates -inadequate respiratory effort or damage to the respiratory centres in the brain, aspiration of amniotic fluid or meconium
**In 'barkers'- defective production of surfactant by the Type 2 alveolar epithelium
*Foals that survive may have hypoxic damage to their brain, walk aimlessly and lack normal sense of fear - "wanderers"
[[Category:Lungs - Developmental Pathology]]
[[Category:Respiratory System - Developmental Pathology]]
*Failure of lung tissue to expand at birth
*Ranges from total in stillborn animals to patchy areas in the case of weak neonates, showing more in the dependant lobes in recumbency
*'''The neonatal respiratory syndrome'''
**Best documented in foals but reported in other species
**Also features extensive atelectasis
**These animals are called 'barkers' due to the noise made during forced expiration
*Gross pathology:
**Red fleshy appearance to the affected part, depressed below the surface if there are adjacent normal aerated areas
**Cut surface oozes blood, perhaps some froth in 'barkers'
**Affected areas sink or are submerged in fixative
*Micro pathology:
**Close approximation of capillary walls
**Little normal alveolar pattern
**'Barkers' may have a proteinaceous fluid in alveoli with hyaline membranes in lower airways
*Cause:
**In weak neonates -inadequate respiratory effort or damage to the respiratory centres in the brain, aspiration of amniotic fluid or meconium
**In 'barkers'- defective production of surfactant by the Type 2 alveolar epithelium
*Foals that survive may have hypoxic damage to their brain, walk aimlessly and lack normal sense of fear - "wanderers"
[[Category:Lungs - Developmental Pathology]]
[[Category:Respiratory System - Developmental Pathology]]