787
edits
Changes
no edit summary
The diagnosis can be confirmed using virus isolation on PK15 or primary pig kidney, using samples from infected tonsils, brain or lung.
The diagnosis can be confirmed using virus isolation on PK15 or primary pig kidney, using samples from infected tonsils, brain or lung.
On post mortem all age ranges have corneal opacity which is mainly unilateral. Piglets show evidence of mild pneumonia, distended bladder and stomach, fibrin strands in the peritoneal cavity and congestion in the brain. Growers show kidney and pericardial haemorrhages aswell as brain congestion.
During infection lymphocytosis and monocytosis may be observed.
On post mortem all age ranges have corneal opacity which is mainly unilateral. Piglets show evidence of mild pneumonia, distended bladder and stomach, fibrin strands in the peritoneal cavity and congestion in the brain. Growers show kidney and pericardial haemorrhages aswell as brain congestion. Necrosis of the seminiferous tubules and rupture of the epidydimis walls and subsequent abscess and granuloma formation has been documented in boars.
'''Differential diagnosis''':PRRS and Pseudorabies.
'''Differential diagnosis''':PRRS and Pseudorabies.
==Treatment==
There is no treatment for blue eye disease. Medication should be used to control the secondary and associated diseases, which are usually respiratory diseases.
==Control==
There is not an approved vaccine for the control of blue eye disease. An experimental killed-virus vaccine was shown to be effective in preventing or minimising the clinical signs (Stephano et al., 1992).
Preventative health programmes that prevent the entrance of the disease are the most effective method of control. Eliminating blue eye disease from affected herds has been achieved with management practices such as closing the herd to the entrance of new susceptible animals, all-in/all-out systems, washing and disinfecting. The disease appears to be self-limiting if properly managed.
==References==
==References==