Changes

Affected animals should be removed from pasture suspected to contain the photodynamic agent. Any drugs given at the time of the condition developing should be stopped. Affected animals should only be allowed access to grazing at night. Anti-inflammatory drugs such as flunixin meglumine and topical steroid creams may be useful to decrease the severity of cutaneous lesions in the acute stage of the disease. Secondary bacterial infections of cutaneous lesions are common and should be treated with antimicrobials and correct wound management. Lesions generally heal well, and surgical debridement is reserved for only the most severly necrotic lesions.

Affected animals should be removed from pasture suspected to contain the photodynamic agent. Any drugs given at the time of the condition developing should be stopped. Affected animals should only be allowed access to grazing at night. Anti-inflammatory drugs such as flunixin meglumine and topical steroid creams may be useful to decrease the severity of cutaneous lesions in the acute stage of the disease. Secondary bacterial infections of cutaneous lesions are common and should be treated with antimicrobials and correct wound management. Lesions generally heal well, and surgical debridement is reserved for only the most severly necrotic lesions.

If the animal is affected by secondary photosensitisation supportive treatment should be provided. A low-protein, high-energy diet rich in branched-chain amino acids such as beet pulp is recommended. Intravenous fluid therapy with 5% dextrose may be necessary in hypoglycaemic patients with signs of hepatic encephalopathy. Severely affected animals with liver failure carry a grave prognosis and are not usually treatable.

If the animal is affected by secondary photosensitisation supportive treatment should be provided. A low-protein, high-energy diet rich in branched-chain amino acids such as beet pulp is recommended. Intravenous fluid therapy with 5% dextrose may be necessary in hypoglycaemic patients with signs of hepatic encephalopathy.  

==Prognosis==

==Prognosis==

In general the prognosis is favourable for primary photosensitisation and poor for secondary photosensitisation due to hepatic involvement. Cutaneous lesions usually resolve within 48 hours but occasionally may persist for up to six months.

In general the prognosis is favourable for primary photosensitisation. Cutaneous lesions usually resolve within 48 hours but occasionally may persist for up to six months. The prognosis is poor for secondary photosensitisation due to hepatic involvement. Severely affected animals with liver failure carry a grave prognosis and are not usually treatable.

==Prevention==

==Prevention==