Changes

The mechanism of  clinical disease in chronic toxoplasmosis is not fully understood, but may be related to low-level tachyzoite replication, or intermittent antigenaemia and parasitaemia^a have been detected in experimentally

The mechanism of  clinical disease in chronic toxoplasmosis is not fully understood, but may be related to low-level tachyzoite replication, or intermittent antigenaemia and parasitemia² have been detected in experimentally

inoculated cats (Burney and others 1999).

inoculated cats (Burney and others 1999). The pathogenesis of disease could also be associated with immunological reactions against the organism through formation and deposition of immune complexes, and delayed hypersensitivity reactions⁴.

is occurring (Lappin and others 1992b, 1996). The

organism is rarely documented in ocular tissues of cats

without fatal disease, yet not all cats with T gondii in

relate to immunological reactions against the organism;

immune complex formation and deposition in tissues and

delayed hypersensitivity reactions may be involved.

ocular toxoplasmosis (Lappin and others 1997).

==Signalment==

==Signalment==