Changes

==Description==

==Description==

'''Caecal impaction''' in horses occurs in two forms. In '''primary impactions''' the caecum contains dry, relatively solid ingesta which may be localised to the cranial part of the caecal base, or extend to fill the whole viscera. The onset of clinical signs is typically 5-7 days and the associated signs are of low grade intermittent colic.  

'''Caecal impaction''' in horses occurs in two forms. In '''primary impactions''' the caecum contains dry, relatively solid ingesta which may be localised to the cranial part of the caecal base, or extend to fill the whole viscera. The onset of [[Colic Diagnosis - Clinical Signs|clinical signs]] is typically 5-7 days and the associated signs are of low grade intermittent colic.

 

'''Secondary impactions''' are a result of caecal dysfunction; the caecal content is fluid and the viscera is distended and immotile. This syndrome usually occurs in the post-operative period and early signs may be missed, being attributed to discomfort following surgery or the concurrent condition. The delayed diagnosis results in a serious situation where the caecum can become so distended that rupture is imminent.

==Signalment==

==Signalment==

Hospitalised horses are at greater risk, especially those with musculoskeletal conditions, often due to inadequate analgesia, the reduction in activity and treatment with non-steroidal anti-inflammatory drugs.

Hospitalised horses are at greater risk, especially those with musculoskeletal conditions, often due to inadequate analgesia, the reduction in activity and treatment with non-steroidal anti-inflammatory drugs.