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*frenzy in response to sensory stimulation
*frenzy in response to sensory stimulation
*conscious proprioceptive deficits
*conscious proprioceptive deficits
With progression, worsening cerebral cortical and cranial nerve dysfunction may result in:
*head pressing
*propulsive walking
*blindness
*circling
*head tilt
*facial and appendicular muscle fasciculations
*paralysis of pharynx, larynx and tongue
*recumbency for 1-7 days followed by death
VEE may have similar or different clinical presentations to WEE and EEE, which may relate to a persistently hightitre viraemia with VEE and differences in strain pathogenicity:
*pyrexia peaks early and remains high throuhgout the disease course
*mild fever and leukopenia associated experimentally with endemic strains
*severe pyrexia and leukopenia associated with epidemic strains
*diarrhoea, severe depression recumbency and death may precede neurological signs
*neurological signs around 4 days post-infection
*other associated signs: abortion, oral ulceration, pulmonary haemorrhage, epistaxis
*Paralysis of the lips
*Paralysis of the lips
====Prognosis====
====Prognosis====
Often '''fatal'''
Often '''fatal''', comatose animals rarely survive. Survivors exhibit functional improvement over weeks to months.
====Control====
====Control====