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Initially, a partial obstruction results.  Overtime, this progresses to a complete obstruction, with obstruction of venous return, arterial occlusion and avulsion of vessels.  The intestinal walls become oedematous, ischaemic and turgid, resulting in devitalisation if not treated.  Adhesion can occur in long standing cases due to fibrin deposition.
 
Initially, a partial obstruction results.  Overtime, this progresses to a complete obstruction, with obstruction of venous return, arterial occlusion and avulsion of vessels.  The intestinal walls become oedematous, ischaemic and turgid, resulting in devitalisation if not treated.  Adhesion can occur in long standing cases due to fibrin deposition.
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===Pathogenesis===
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[[Image:intussuceptionphoto.jpg|thumb|right|150px|Intersusception (Courtesy of Bristol BioMed Image Archive)]]
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The proximal intestine invaginates into the adjacent distal portion, taking its mesenteric attachment with it. Compression of the mesenteric vessels obstructs venous drainage of the gut, resulting in venous congestion, leading to swelling and oedema.
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An inflammatory exudate is released from the serosal surface, fibrinous adhesions may form between surfaces making the structure irreducible this may progress to necrosis of the tissue.
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There is functional obstruction to bowel.
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May rupture, leading to peritonitis and death.
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*Associated with any condition that increases peristalsis
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** Enteritis
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** Foreign body
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** Heavy parasitism
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** Previous intestinal surgery
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** Intramural abscess/tumour
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** Motility disorders.
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** Change in diet
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** Bacterial infection
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==Signalment==
 
==Signalment==
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====Pathogenesis====
  −
  −
[[Image:intussuceptionphoto.jpg|thumb|right|150px|Intersusception (Courtesy of Bristol BioMed Image Archive)]]
  −
The proximal intestine invaginates into the adjacent distal portion, taking its mesenteric attachment with it. Compression of the mesenteric vessels obstructs venous drainage of the gut, resulting in venous congestion, leading to swelling and oedema.
  −
An inflammatory exudate is released from the serosal surface, fibrinous adhesions may form between surfaces making the structure irreducible this may progress to necrosis of the tissue.
  −
There is functional obstruction to bowel.
  −
May rupture, leading to peritonitis and death.
  −
*Associated with any condition that increases peristalsis
  −
** Enteritis
  −
** Foreign body
  −
** Heavy parasitism
  −
** Previous intestinal surgery
  −
** Intramural abscess/tumour
  −
** Motility disorders.
  −
** Change in diet
  −
** Bacterial infection
     −
====Pathology====
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===Pathology===
 
The degree of damage to the intestine depends on the severity of the intussusception. In severe or chronic cases fibrinous adhesions form between surfaces making the structure irreducible. Necrosis of the tissue usually follows.
 
The degree of damage to the intestine depends on the severity of the intussusception. In severe or chronic cases fibrinous adhesions form between surfaces making the structure irreducible. Necrosis of the tissue usually follows.
  
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