Humoral immunity includes [[Complement|Complement]] activation of the classical pathway. It results in the production of [[Immunoglobulin M|IgM]] and [[Immunoglobulin G|IgG]] and makes the complement system more efficient.
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Humoral immunity includes [[Complement|complement]] activation of the classical pathway. It results in the production of [[Immunoglobulin M|IgM]] and [[Immunoglobulin G|IgG]] and makes the complement system more efficient.
==Cell-Mediated==
==Cell-Mediated==
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==Extracellular Infection==
==Extracellular Infection==
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The response to extracellular infection involves [[Complement|Complement]] and phagocytosis; [[Lymphocytes#B Cells|B cell]] and T helper type II cell stimulation and the production of [[Immunoglobulin M|IgM]], which activates the classical cascade. There is also class switching of [[Immunoglobulin M|IgM]] to [[Immunoglobulin G|IgG]], which is a good opsonin and targets bacterial Fcγ receptor expressed by [[Macrophages|macrophages]] and [[Neutrophils|neutrophils]].
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The response to extracellular infection involves [[Complement|complement]] and phagocytosis; [[Lymphocytes#B Cells|B cell]] and T helper type II cell stimulation and the production of [[Immunoglobulin M|IgM]], which activates the classical cascade. There is also class switching of [[Immunoglobulin M|IgM]] to [[Immunoglobulin G|IgG]], which is a good opsonin and targets bacterial Fcγ receptor expressed by [[Macrophages|macrophages]] and [[Neutrophils|neutrophils]].
==Vesicular Infection==
==Vesicular Infection==
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[[Category:To Do - AimeeHicks]] [[Category:To Do - AP Review]]