Ascaridia can be diagnosed by the above clinical, faecal examination or by post-mortem (PM). Evidence of enteritis/haemorrhagic enteritis can be seen on PM; as large numbers of larvae in the histotropic phase can cause extensive damage to the glandular epithelium. Adhesion of the mucosal villi can also be noted as a result of the proliferation of mucous-secretory cells.
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*Clinical signs
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*Faecal examination
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*Post-mortem (PM)- evidence of enteritis/haemorrhagic enteritis, extensive damage to the glandular epithelium, adhesion of the mucosal villi and pressure atrophy from adult worms.
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Adult worms can also cause damage to the epithelia as a result of pressure atrophy of the villi, causing occasional necrosis of the mucosal layer. In chronic infections the intestinal wall can become distended as muscle tone is lost.
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In chronic infections the intestinal wall can become distended as muscle tone is lost.
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|l18=Ascaridia - Poultry#Diagnosis
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|q19=How can ''A.galli'' be treated?
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|a19=
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In feed treatment with either:
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*Piperazine salts
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*Levamisole
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*Benzimidazole
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|l19=Ascaridia - Poultry#Treatment
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|q20=How can you control ''ascaridia'' infections?
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|a20=
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*Free range systems, young birds can be iolated and put onto ground previously unused by poultry
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*Rotation of poultry runs
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*Raised feeding and watering stations - reducing faecal-oral transmission