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The other granulocytes present in parasite-infected inflammatory lesions and involved in the anti-parasitic response are the '''[[Neutrophils|neutrophils]]'''. In the parasitic response they have similar properties to macrophages with activation caused by cytokines such as TNFα, IFNγ and GM-CSF. Their mechanism for killing is by an intense respiratory burst, with extracellular killing being mediated by hydrogen peroxide (H<sub>2</sub>O<sub>2</sub>). In addition, they also express Fc and complement receptors so can participate in antibody-dependent cell-mediated cytotoxicity and [[Phagocytosis|phagocytosis]].
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The other granulocytes present in parasite-infected inflammatory lesions and involved in the anti-parasitic response are the '''[[Neutrophils|neutrophils]]'''. In the parasitic response they have similar properties to macrophages with activation caused by cytokines such as TNFα, IFNγ and GM-CSF. Their mechanism for killing is by an intense respiratory burst, with extracellular killing being mediated by hydrogen peroxide (H<sub>2</sub>O<sub>2</sub>). In addition, they also express Fc and [[Complement|complement]] receptors so can participate in antibody-dependent cell-mediated cytotoxicity and [[Phagocytosis|phagocytosis]].
 
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