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| **Beta toxin plays major role in pathogenesis of the disease | | **Beta toxin plays major role in pathogenesis of the disease |
| **Post mortem: jejunal ulceration; hyperaemia in small intestine; fluid accumulation in peritoneal cavity; congestion of peritoneal vessels; petechial haemorrhages | | **Post mortem: jejunal ulceration; hyperaemia in small intestine; fluid accumulation in peritoneal cavity; congestion of peritoneal vessels; petechial haemorrhages |
− | ** | + | **Haemorrhagic enteritis in piglets |
| + | ***Peracute enterotoxaemia often of entire litter with mortality rates 80% |
| + | ***Infection from sow's faeces |
| + | ***Death within 24 hours in young piglets |
| + | ***Chronic disease in older piglets |
| + | ***Dullness, anorexia, bloody faeces, perianal hyperaemia |
| + | ***Post mortem: necrosis of terminal small intestinal mucosa, caecum and colon and blood-stained contents; serosanguinous fluid in pleural and peritoneal cavities |
| + | **Necrotic enteritis in chickens: |
| + | ***Broilers under 12 weeks |
| + | ***Acute enterotoxaemia, sudden onset and high mortality |
| + | ***Necrosis of small intestine |
| + | ***Predisposing factors include diet changes, coccidial infection and intestinal hypomotility |
| *''C. perfringens'' type D: | | *''C. perfringens'' type D: |
| **[[Intestines - Catarrhal Enteritis#"Pulpy Kidney" Disease|Pulpy kidney disease]] in well-fed 3-10 week-old lambs | | **[[Intestines - Catarrhal Enteritis#"Pulpy Kidney" Disease|Pulpy kidney disease]] in well-fed 3-10 week-old lambs |