Changes

*Hypovolaemic shock secondary to the rapid lysis of red blood cells and their functional masses as vascular expanders. <br><br>

*Hypovolaemic shock secondary to the rapid lysis of red blood cells and their functional masses as vascular expanders. <br><br>

*Loss of oxygen carrying capacity from the lysed red blood cells. <br><br>

*Loss of oxygen carrying capacity from the lysed red blood cells. <br><br>

*The development of kernicterus. The increasingly elevated levels of bilirubin with in the bloodstream can cross the immature blood–brain barrier in the foal and result in chemical toxicity to the brain. The brain damage can be permanent and be represented by persistent seizures or maladjustment.

*The development of kernicterus. The increasingly elevated levels of bilirubin within the bloodstream can cross the immature blood–brain barrier in the foal and result in chemical toxicity to the brain. The brain damage can be permanent and be represented by persistent seizures or maladjustment.

|l2=Neonatal Isoerythrolysis#Clinical Signs

|l2=Neonatal Isoerythrolysis#Clinical Signs

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