Changes

The three possible areas for aortic stenosis to occur are '''subvalvular or subaortic''' (occurs below the valve), '''valvular or aortic''' (occurs at the valve) and '''supravalvular''' (occurs after the valve). The most common form is subaortic stenosis (SAS), usually as a consequence of fibrous bands that may be circumferential (severe) or may only affect a small area of the LVOTO. The subvalvular fibrous ring may encompass the anterior mitral valve leaflet, causing it to become immobile.  In subaortic stenosis, the lesions may not be present from birth and may develop or progress as the dog matures; but do not generally progress beyond early adulthood. This is in contrast to valvular aortic stenosis, where the valve may become more stenotic as the dog ages.  

The three possible areas for aortic stenosis to occur are '''subvalvular or subaortic''' (occurs below the valve), '''valvular or aortic''' (occurs at the valve) and '''supravalvular''' (occurs after the valve). The most common form is subaortic stenosis (SAS), usually as a consequence of fibrous bands that may be circumferential (severe) or may only affect a small area of the LVOTO. The subvalvular fibrous ring may encompass the anterior mitral valve leaflet, causing it to become immobile.  In subaortic stenosis, the lesions may not be present from birth and may develop or progress as the dog matures; but do not generally progress beyond early adulthood. This is in contrast to valvular aortic stenosis, where the valve may become more stenotic as the dog ages.  

Aortic stenosis causes an impedence to left ventricular ejection and consequent pressure overload of the left ventricle. The increase in afterload results in a compensatory ''''concentric left ventricular hypertrophy'''' in attempt to maintain sufficient cardiac output. The severity of stenosis usually determines the extent of hypertrophy. The myocardial hypertrophy results in increased myocardial oxygen demand, beyond the capacity of the coronary capillary vascular bed to supply oxygen. Increased wall stress also compromises coronary perfusion, particularly to the subendocardium. Insufficient oxygen supply to meet the needs of the hypertrophied left ventricle causes myocardial ischaemia and predisposes the animal to ventricular [[:Category:Arrhythmia|arrhythmias]]. There is also an increased risk of bacterial [[Endocarditis|endocarditis]] in animals affected by this condition.

Aortic stenosis causes an impedence to left ventricular ejection and consequent pressure overload of the left ventricle. The increase in afterload results in a compensatory ''''concentric left ventricular hypertrophy'''' in attempt to maintain sufficient cardiac output. The severity of stenosis usually determines the extent of hypertrophy. The myocardial hypertrophy results in increased myocardial oxygen demand, beyond the capacity of the coronary capillary vascular bed to supply oxygen. Increased wall stress also compromises coronary perfusion, particularly to the subendocardium. Insufficient oxygen supply to meet the needs of the hypertrophied left ventricle causes myocardial ischaemia and predisposes the animal to ventricular [[:Category:Arrhythmia|arrhythmias]].  

 

There is also an increased risk of bacterial [[Endocarditis|endocarditis]] in animals affected by this condition.

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