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'''''Aetiology'''''
<big><center>[[Reproductive System|'''BACK TO REPRODUCTIVE SYSTEM''']]</center></big>
<big><center>[[Reproductive_System#Fertilisation.2C_Implantation_and_Early_Embryonic_Development|'''BACK TO FERTILISATION , IMPLANTATION and EARLY EMBRYONIC DEVELOPMENT''']]</center></big>
<big><center>[[Fertilisation%2C_Implantation_and_Early_Embryonic_Development_-Sexual_Differentiation-_Anatomy_%26_Physiology#Associated_Disorders|'''BACK TO SEXUAL DIFFERENTIATION''']]</center></big>
== Aetiology ==
* Genotype is XX as should be female
* Genotype is XX as should be female
* Ovaries develop normally
* Ovaries develop normally
* Foetal [[Endocrine_System_-_Adrenal_Glands_-_Anatomy_%26_Physiology|Adrenal Gland]] is hyperactive and secretes large quantities of Adrenal Androgens, due to a '''21 hydroxylase mutation'''.
* Foetal [[Endocrine_System_-_Adrenal_Glands_-_Anatomy_%26_Physiology|Adrenal Gland]] is hyperactive and secretes large quantities of Adrenal Androgens, due to a '''21 hydroxylase mutation'''.
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== Consequence ==
* Wolffian Ducts persist due to the presence of Androgens, thus allowing development of '''male internal and external genitalia'''.
* Wolffian Ducts persist due to the presence of Androgens, thus allowing development of '''male internal and external genitalia'''.
* Mullarian Ducts persist due to the absence of testes, hence absence of Sertoli cell MIH secretion. This allows formation of '''female internal genitalia'''.
* Mullarian Ducts persist due to the absence of testes, hence absence of Sertoli cell MIH secretion. This allows formation of '''female internal genitalia'''.
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== Morphology ==
* Appears male with a '''micropenis and scrotum'''
* Appears male with a '''micropenis and scrotum'''
* Both male and female internal genitalia
* Both male and female internal genitalia