Changes

== Introduction ==

== Introduction ==

Cholesterol, the most common steroid in the body, is derived from the diet and synthesised in the liver. Cholesterol is the precursor of steroid hormones and can be degraded to bile acids, and like bile acids undergoes enterohepatic recycling. As a marker of liver function it has limited value as levels may be increased, decreased or normal according to dietary intake and the underlying disease process. Fluctuations in serum cholesterol are considered to be minimal even after feeding a high fat diet and persistently elevated levels particularly in fasted animals warrant investigation. Hypercholesterolaemia in the absence of hypertriglyceridaemia does not result in sample lipaemia.

Cholesterol, the most common steroid in the body, is derived from the diet and synthesised in the liver. Cholesterol is the precursor of steroid hormones and can be degraded to bile acids, and like bile acids undergoes enterohepatic recycling. As a marker of liver function it has limited value as levels may be increased, decreased or normal according to dietary intake and the underlying disease process. Fluctuations in serum cholesterol are considered to be minimal even after feeding a high fat diet and persistently elevated levels particularly in fasted animals warrant investigation. Hypercholesterolaemia in the absence of hypertriglyceridaemia does not result in sample lipaemia.