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| + | '''Use the mouse to highlight the answers. They are written in white''' |
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| <TR> | | <TR> |
− | <TD>What are the problems if body water levels drop too low?</TD> | + | <TD>What the effects of varying sodium concentration within the body?</TD> |
| <TD><FONT COLOR="#FFFFFF"> | | <TD><FONT COLOR="#FFFFFF"> |
− | * Inadequate perfusion due to low blood volume | + | *Too low |
− | * A build up of waste products due to insufficient urine volume | + | **Water and ECF volumes fall |
− | * Increased solute concentrations affect cell function | + | **Sodium dependant transporters are disturbed |
− | * Inability to sweat and control body temperature | + | *Too High |
− | * Death</font></TD> | + | **Water and ECF volume increases |
− | <TD>[[Water Balance and Homeostasis - Physiology#Too Little Water|Link]]</TD> | + | **Sodium dependant transporters are disturbed </font></TD> |
| + | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#Sodium|Link]]</TD> |
| </TR> | | </TR> |
| + | <TR> |
| + | <TD>Why is sodium not regulated on the basis of plasma concentration?</TD> |
| + | <TD><FONT COLOR="#FFFFFF"> |
| + | *Water follows sodium. Therefore volume notconcentration alter |
| + | *The hormonal controllers affect sodium concentration and ECF concentration |
| + | *ADH and the thirst response dilute any increase in sodium by increasing ECF volume</font></TD> |
| + | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#Regulation|Link]]</TD> |
| + | </TR> |
| + | <TR> |
| + | <TD>What is "salt hunger"?</TD> |
| + | <TD><FONT COLOR="#FFFFFF"> |
| + | It's where an animal with low body salt concentrations actually craves salt</font></TD> |
| + | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#Salt Hunger|Link]]</TD> |
| + | </TR> |
| + | <TR> |
| + | <TD>What are the three major sources of potassium?</TD> |
| + | <TD><FONT COLOR="#FFFFFF"> |
| + | * Diffusion from the small intestine |
| + | * Active transport from the colon |
| + | * Recovered from cellular breakdown</font></TD> |
| + | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#Sources|Link]]</TD> |
| + | </TR> |
| + | <TR> |
| + | <TD>By which three methods is potassium homeostasis managed? Which of these make up the chronic and which the acute response</TD> |
| + | <TD><FONT COLOR="#FFFFFF"> |
| + | *Cellular translocation - Acute |
| + | *Renal excretion - 90% of chronic |
| + | *GI excretion - 10% of chronic</font></TD> |
| + | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#Methods of Control|Link]]</TD> |
| + | </TR> |
| + | <TR> |
| + | <TD>Which two things control the acute response and how do they do it?</TD> |
| + | <TD><FONT COLOR="#FFFFFF"> |
| + | *Insulin |
| + | *Activation of beta2 adrenoreceptors |
| + | *Work by increasing the activity of Na+ / K+ ATPases causing sodium efflux and potassium influx</font></TD> |
| + | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#|Link]]</TD> |
| + | </TR> |
| + | <TR> |
| + | <TD>?</TD> |
| + | <TD><FONT COLOR="#FFFFFF"> |
| + | </font></TD> |
| + | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#|Link]]</TD> |
| + | </TR> |
| + | <TR> |
| + | <TD>?</TD> |
| + | <TD><FONT COLOR="#FFFFFF"> |
| + | </font></TD> |
| + | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#|Link]]</TD> |
| + | </TR> |
| + | <TR> |
| + | <TD>?</TD> |
| + | <TD><FONT COLOR="#FFFFFF"> |
| + | </font></TD> |
| + | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#|Link]]</TD> |
| + | </TR> |
| + | <TR> |
| + | <TD>?</TD> |
| + | <TD><FONT COLOR="#FFFFFF"> |
| + | </font></TD> |
| + | <TD>[[Essential Ion and Compound Balance and Homeostasis - Anatomy & Physiology#|Link]]</TD> |
| + | </TR> |
| + | </table> |