Changes

=Immunity to fungi=

=Immunity to fungi=

Cutaneous and superficial fungal infections are normally self-limiting, with recovery associated with a resistance to re-infection. As hosts often develop delayed-type hypersensitivity, this resistance appears to be cell-mediated.

Cutaneous and superficial fungal infections are normally self-limiting, with recovery associated with a resistance to re-infection. As hosts often develop delayed-type hypersensitivity, this resistance appears to be cell-mediated.

*T cell immunity- it is thought that helper T cells release cytokines to activate macrophages to fight the infection

*[[T cell differentiation - WikiBlood|T cell]] immunity- it is thought that [[T cell differentiation - WikiBlood#TH1 Cells|helper T cells]] release cytokines to activate [[Macrophages - WikiBlood|macrophages]] to fight the infection

*Neutrophils- there is now growing evidence for the role of neutrophils in some infections of the respiratory system, with defensin and the nitric oxide pathways implicated

*[[Neutrophils - WikiBlood|Neutrophils]]- there is now growing evidence for the role of [[Neutrophils - WikiBlood|neutrophils]] in some infections of the respiratory system, with defensin and the nitric oxide pathways implicated