Changes

choice4="GLUT 4"

choice4="GLUT 4"

correctchoice="5"

correctchoice="5"

feedback5="'''Correct!''' GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and Absorption|WikiVet Article: carbohydrate digestion and absorption]]."

feedback5="'''Correct!''' GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and Absorption|WikiVet Article: Carbohydrate digestion and absorption]]"

feedback3="'''Incorrect.''' The glucose/Na+ symport is responsible for the uptake of glucose from the intestinal lumen into intestinal cells. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and Absorption|WikiVet Article: carbohydrate digestion and absorption]]."

feedback3="'''Incorrect.''' The glucose/Na+ symport is responsible for the uptake of glucose from the intestinal lumen into intestinal cells. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and Absorption|WikiVet Article: Carbohydrate digestion and absorption]]"

feedback2="'''Incorrect.''' The Na+/K+ ATPase is responsible for pumping sodium ions into the blood in order to maintain a low concentration of sodium inside the intestinal cells. This is important as the action of the glucose/Na+ symport depends upon their being a lower concentration of sodium inside the intestinal cells than in the gut lumen. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and Absorption|WikiVet Article: carbohydrate digestion and absorption]]."

feedback2="'''Incorrect.''' The Na+/K+ ATPase is responsible for pumping sodium ions into the blood in order to maintain a low concentration of sodium inside the intestinal cells. This is important as the action of the glucose/Na+ symport depends upon their being a lower concentration of sodium inside the intestinal cells than in the gut lumen. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and Absorption|WikiVet Article: Carbohydrate digestion and absorption]]"

feedback1="'''Incorrect.''' γ Glutamyl transferase spans the enterocyte membrane and combines glutathione from the inside of the cell with a di-,tri- or oligo-peptide from the intestinal lumen forming a γ-glu-aa complex which is transported into the cell. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and Absorption |WikiVet Article: carbohydrate digestion and absorption]]."

feedback1="'''Incorrect.''' γ Glutamyl transferase spans the enterocyte membrane and combines glutathione from the inside of the cell with a di-,tri- or oligo-peptide from the intestinal lumen forming a γ-glu-aa complex which is transported into the cell. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and Absorption |WikiVet Article: Carbohydrate digestion and absorption]]"

feedback4="'''Incorrect.''' GLUT 4 transporters are used for uptake of glucose into muscle and adipose tissue cells. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and AbsorptionPancreas - Anatomy & Physiology|WikiVet Article: carbohydrate digestion and absorption, pancreas]]."

feedback4="'''Incorrect.''' GLUT 4 transporters are used for uptake of glucose into muscle and adipose tissue cells. GLUT 5 transporters are responsible for uptake of glucose from intestinal cells into the blood. [[Small Intestine - Anatomy & Physiology#Carbohydrate Digestion and AbsorptionPancreas - Anatomy & Physiology|WikiVet Article: Carbohydrate digestion and absorption]]"

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</WikiQuiz>

</WikiQuiz>

choice5="Somatostatin, Dopamine and Oxytocin"

choice5="Somatostatin, Dopamine and Oxytocin"

correctchoice="4"

correctchoice="4"

feedback4="'''Correct!''' Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary Gland - Anatomy & Physiology|WikiVet Article: pituitary gland]]."

feedback4="'''Correct!''' Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary Gland - Anatomy & Physiology|WikiVet Article: Pituitary gland]]"

feedback1="'''Incorrect.''' Prolactin is secreted by the anterior pituitary and dopamine is secreted by the hypothalamus. Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary Gland - Anatomy & PhysiologyHypothalamus - Anatomy & Physiology|WikiVet Article: pituitary gland]]."

feedback1="'''Incorrect.''' Prolactin is secreted by the anterior pituitary and dopamine is secreted by the hypothalamus. Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary Gland - Anatomy & PhysiologyHypothalamus - Anatomy & Physiology|WikiVet Article: Pituitary gland]]"

feedback3="'''Incorrect.''' Prolactin is secreted by the anterior pituitary and somatostatin is secreted by the hypothalamus. Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary Gland - Anatomy & PhysiologyHypothalamus - Anatomy & Physiology|WikiVet Article: pituitary gland]]."

feedback3="'''Incorrect.''' Prolactin is secreted by the anterior pituitary and somatostatin is secreted by the hypothalamus. Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary Gland - Anatomy & PhysiologyHypothalamus - Anatomy & Physiology|WikiVet Article: Pituitary gland]]"

feedback2="'''Incorrect.''' Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. Dopamine is synthesised in several areas of the brain, including the hypothalamus but is not secreted by the posterior pituitary. [[Pituitary Gland - Anatomy & PhysiologyHypothalamus - Anatomy & Physiology|WikiVet Article: pituitary gland]]."

feedback2="'''Incorrect.''' Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. Dopamine is synthesised in several areas of the brain, including the hypothalamus but is not secreted by the posterior pituitary. [[Pituitary Gland - Anatomy & PhysiologyHypothalamus - Anatomy & Physiology|WikiVet Article: Pituitary gland]]"

feedback5="'''Incorrect.''' Somatostatin is secreted by the hypothalamus, dopamine is synthesised in several areas of the brain, including the hypothalamus but is not secreted by the posterior pituitary. Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary Gland - Anatomy & PhysiologyHypothalamus - Anatomy & Physiology|WikiVet Article: pituitary gland]]."

feedback5="'''Incorrect.''' Somatostatin is secreted by the hypothalamus, dopamine is synthesised in several areas of the brain, including the hypothalamus but is not secreted by the posterior pituitary. Oxytocin and ADH are produced by cell bodies in the paraventricular and supraoptic nuclei of the hypothalamus. They are then transported down axons into the posterior pituitary for storage, prior to release. [[Pituitary Gland - Anatomy & PhysiologyHypothalamus - Anatomy & Physiology|WikiVet Article: Pituitary gland]]"

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</WikiQuiz>  

</WikiQuiz>  

choice1="The ascending limb of the loop of Henle."

choice1="The ascending limb of the loop of Henle."

correctchoice="2"

correctchoice="2"

feedback2="'''Correct!''' All major hormonal controls of reabsorption are exerted on these parts of the nephron. [[Aldosterone#Aldosterone |WikiVet Article: aldosterone]]."

feedback2="'''Correct!''' All major hormonal controls of reabsorption are exerted on these parts of the nephron. [[Aldosterone#Aldosterone |WikiVet Article: Aldosterone]]"

feedback5="'''Incorrect.''' All major hormonal controls of reabsorption are exerted on the late distal convoluted tubule and the collecting ducts. [[Aldosterone#Aldosterone|WikiVet Article: aldosterone. ]]"

feedback5="'''Incorrect.''' All major hormonal controls of reabsorption are exerted on the late distal convoluted tubule and the collecting ducts. [[Aldosterone#Aldosterone|WikiVet Article: Aldosterone]]"

feedback4="'''Incorrect.''' All major hormonal controls of reabsorption are exerted on the collecting ducts. [[Aldosterone#Aldosterone|WikiVet Article: aldosterone. ]]"

feedback4="'''Incorrect.''' All major hormonal controls of reabsorption are exerted on the collecting ducts. [[Aldosterone#Aldosterone|WikiVet Article: Aldosterone]]"

feedback3="'''Incorrect.''' All major hormonal controls of reabsorption are exerted on the late distal convoluted tubule and the collecting ducts. [[Aldosterone#Aldosterone |WikiVet Article: aldosterone. ]]"

feedback3="'''Incorrect.''' All major hormonal controls of reabsorption are exerted on the late distal convoluted tubule and the collecting ducts. [[Aldosterone#Aldosterone |WikiVet Article: Aldosterone]]"

feedback1="'''Incorrect.''' All major hormonal controls of reabsorption are exerted on the late distal convoluted tubule and the collecting ducts. [[Aldosterone#Aldosterone|WikiVet Article: aldosterone. ]]"

feedback1="'''Incorrect.''' All major hormonal controls of reabsorption are exerted on the late distal convoluted tubule and the collecting ducts. [[Aldosterone#Aldosterone|WikiVet Article: Aldosterone]]"

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</WikiQuiz>

</WikiQuiz>

choice1="Glycogenolysis and glycolysis"

choice1="Glycogenolysis and glycolysis"

correctchoice="5"

correctchoice="5"

feedback5="'''Correct!''' The function of insulin is to reduce blood sugar levels when they rise too high. Therefore glycolysis (glucose breakdown) is stimulated as this process uses glucose to make ATP, NADH and pyruvate. And glycogenesis (glycogen synthesis) is stimulated, as in this pathway glucose is used to make the storage product, glycogen. [[Pancreas - Anatomy & Physiology#Insulin|WikiVet Article: pancreas]]."

feedback5="'''Correct!''' The function of insulin is to reduce blood sugar levels when they rise too high. Therefore glycolysis (glucose breakdown) is stimulated as this process uses glucose to make ATP, NADH and pyruvate. And glycogenesis (glycogen synthesis) is stimulated, as in this pathway glucose is used to make the storage product, glycogen. [[Pancreas - Anatomy & Physiology#Insulin|WikiVet Article: Pancreas]]"

feedback4="'''Incorrect.''' The function of insulin is to reduce blood sugar levels when they rise too high. The process of gluconeogenesis (glucose synthesis) would further increase glucose levels and is therefore not stimulated by high blood glucose. Glycolysis (glucose breakdown) is stimulated as this process uses glucose to make ATP, NADH and pyruvate. Glycogenesis (glycogen synthesis) is also stimulated, as in this pathway glucose is used to make the storage product, glycogen. [[Pancreas - Anatomy & Physiology#Insulin|WikiVet Article: pancreas]]."

feedback4="'''Incorrect.''' The function of insulin is to reduce blood sugar levels when they rise too high. The process of gluconeogenesis (glucose synthesis) would further increase glucose levels and is therefore not stimulated by high blood glucose. Glycolysis (glucose breakdown) is stimulated as this process uses glucose to make ATP, NADH and pyruvate. Glycogenesis (glycogen synthesis) is also stimulated, as in this pathway glucose is used to make the storage product, glycogen. [[Pancreas - Anatomy & Physiology#Insulin|WikiVet Article: Pancreas]]"

feedback3="'''Incorrect.''' The function of insulin is to reduce blood sugar levels when they rise too high.The process of gluconeogenesis (glucose synthesis) would further increase glucose levels and is therefore not stimulated by high blood glucose. Glycolysis (glucose breakdown) is stimulated as this process uses glucose to make ATP, NADH and pyruvate. Glycogenesis (glycogen synthesis) is also stimulated, as in this pathway glucose is used to make the storage product, glycogen. [[Pancreas - Anatomy & Physiology#Insulin|WikiVet Article: pancreas]]."

feedback3="'''Incorrect.''' The function of insulin is to reduce blood sugar levels when they rise too high.The process of gluconeogenesis (glucose synthesis) would further increase glucose levels and is therefore not stimulated by high blood glucose. Glycolysis (glucose breakdown) is stimulated as this process uses glucose to make ATP, NADH and pyruvate. Glycogenesis (glycogen synthesis) is also stimulated, as in this pathway glucose is used to make the storage product, glycogen. [[Pancreas - Anatomy & Physiology#Insulin|WikiVet Article: Pancreas]]"

feedback2="'''Incorrect.''' The function of insulin is to reduce blood sugar levels when they rise too high. Glycogenesis (glycogen synthesis) is stimulated, as in this pathway glucose is used to make the storage product, glycogen. Glycolysis (glucose breakdown) is stimulated as this process uses glucose to make ATP, NADH and pyruvate. Glycogenolysis is the breakdown of glycogen stimulated by glucagon. [[Pancreas - Anatomy & Physiology#Insulin|WikiVet Article: pancreas]]."

feedback2="'''Incorrect.''' The function of insulin is to reduce blood sugar levels when they rise too high. Glycogenesis (glycogen synthesis) is stimulated, as in this pathway glucose is used to make the storage product, glycogen. Glycolysis (glucose breakdown) is stimulated as this process uses glucose to make ATP, NADH and pyruvate. Glycogenolysis is the breakdown of glycogen stimulated by glucagon. [[Pancreas - Anatomy & Physiology#Insulin|WikiVet Article: Pancreas]]"

feedback1="'''Incorrect.''' The function of insulin is to reduce blood sugar levels when they rise too high. Glycogenolysis is not stimulated as this would produce more glucose through glycogen breakdown.This processwould stimulated by glucago. Glycogenesis is stimulated, as in this pathway glucose is used to make the storage product, glycogen. Glycolysis is stimulated as this process uses glucose to make ATP, NADH and pyruvate. [[Pancreas - Anatomy & Physiology#Insulin|WikiVet Article: pancreas]]."

feedback1="'''Incorrect.''' The function of insulin is to reduce blood sugar levels when they rise too high. Glycogenolysis is not stimulated as this would produce more glucose through glycogen breakdown.This processwould stimulated by glucago. Glycogenesis is stimulated, as in this pathway glucose is used to make the storage product, glycogen. Glycolysis is stimulated as this process uses glucose to make ATP, NADH and pyruvate. [[Pancreas - Anatomy & Physiology#Insulin|WikiVet Article: Pancreas]]"

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</WikiQuiz>  

</WikiQuiz>  

choice5="Stimulates glycolysis and and inhibits glycogenesis"

choice5="Stimulates glycolysis and and inhibits glycogenesis"

correctchoice="4"

correctchoice="4"

feedback4="'''Correct!''' Glucagon is released in response to low blood sugar levels in order to stimulate processes that will raise blood glucose concentrations. Therefore glucagon stimulates gluconeogenesis which produces glucose from non-carbohydrate sources and also stimulates glycogenolysis, which is the breakdown of stored glycogen to glucose. [[Pancreas - Anatomy & Physiology#Glucagon|WikiVet Article: pancreas]]."

feedback4="'''Correct!''' Glucagon is released in response to low blood sugar levels in order to stimulate processes that will raise blood glucose concentrations. Therefore glucagon stimulates gluconeogenesis which produces glucose from non-carbohydrate sources and also stimulates glycogenolysis, which is the breakdown of stored glycogen to glucose. [[Pancreas - Anatomy & Physiology#Glucagon|WikiVet Article: Pancreas]]"

feedback2="'''Incorrect.''' Glucagon is released in response to low blood sugar levels in order to stimulate processes that will raise serum glucose concentrations. Therefore glucagon stimulates gluconeogenesis which produces glucose from non-carbohydrate sources and also stimulates glycogenolysis, which is the breakdown of stored glycogen to glucose. Glycolysis (glucose breakdown) is not stimulated, this would lead to the breakdown of glucose, further reducing blood levels. [[Pancreas - Anatomy & Physiology#Glucagon|WikiVet Article: pancreas]]."

feedback2="'''Incorrect.''' Glucagon is released in response to low blood sugar levels in order to stimulate processes that will raise serum glucose concentrations. Therefore glucagon stimulates gluconeogenesis which produces glucose from non-carbohydrate sources and also stimulates glycogenolysis, which is the breakdown of stored glycogen to glucose. Glycolysis (glucose breakdown) is not stimulated, this would lead to the breakdown of glucose, further reducing blood levels. [[Pancreas - Anatomy & Physiology#Glucagon|WikiVet Article: Pancreas]]"

feedback1="'''Incorrect.''' Glucagon is released in response to low blood sugar levels in order to stimulate processes that will raise serum glucose concentrations. Therefore glucagon stimulates gluconeogenesis which produces glucose from non-carbohydrate sources and also stimulates glycogenolysis, which is the breakdown of stored glycogen to glucose. [[Pancreas - Anatomy & Physiology#Glucagon|WikiVet Article: pancreas]]."

feedback1="'''Incorrect.''' Glucagon is released in response to low blood sugar levels in order to stimulate processes that will raise serum glucose concentrations. Therefore glucagon stimulates gluconeogenesis which produces glucose from non-carbohydrate sources and also stimulates glycogenolysis, which is the breakdown of stored glycogen to glucose. [[Pancreas - Anatomy & Physiology#Glucagon|WikiVet Article: Pancreas]]"

feedback3="'''Incorrect.''' Glucagon is released in response to low blood sugar levels in order to stimulate processes that will raise serum glucose concentrations. Therefore you are correct that glucagon stimulates gluconeogenesis (glucose synthesis) which produces glucose from non-carbohydrate sources, but glucagon also stimulates glycogenolysis, which is the breakdown of stored glycogen to glucose. [[Pancreas - Anatomy & Physiology#Glucagon|WikiVet Article: pancreas]]."

feedback3="'''Incorrect.''' Glucagon is released in response to low blood sugar levels in order to stimulate processes that will raise serum glucose concentrations. Therefore you are correct that glucagon stimulates gluconeogenesis (glucose synthesis) which produces glucose from non-carbohydrate sources, but glucagon also stimulates glycogenolysis, which is the breakdown of stored glycogen to glucose. [[Pancreas - Anatomy & Physiology#Glucagon|WikiVet Article: Pancreas]]"

feedback5="'''Incorrect.''' Glucagon is released in response to low blood sugar levels in order to stimulate processes that will raise serum glucose concentrations. Therefore glucagon stimulates gluconeogenesis (glucose synthesis) which produces glucose from non-carbohydrate sources and also stimulates glycogenolysis, which is the breakdown of stored glycogen to glucose. [[Pancreas - Anatomy & Physiology#Glucagon|WikiVet Article: pancreas]]."

feedback5="'''Incorrect.''' Glucagon is released in response to low blood sugar levels in order to stimulate processes that will raise serum glucose concentrations. Therefore glucagon stimulates gluconeogenesis (glucose synthesis) which produces glucose from non-carbohydrate sources and also stimulates glycogenolysis, which is the breakdown of stored glycogen to glucose. [[Pancreas - Anatomy & Physiology#Glucagon|WikiVet Article: Pancreas]]"

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</WikiQuiz>  

</WikiQuiz>  

choice4="PTH increases renal excretion of calcitriol."

choice4="PTH increases renal excretion of calcitriol."

correctchoice="3"

correctchoice="3"

feedback3="'''Correct!''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: calcium homeostasis]]."

feedback3="'''Correct!''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: Calcium homeostasis]]"

feedback5="'''Incorrect.''' The 25- hydroxylation reaction in the liver is the unregulated step that produces calcidiol. PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: calcium homeostasis]]."

feedback5="'''Incorrect.''' The 25- hydroxylation reaction in the liver is the unregulated step that produces calcidiol. PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: Calcium homeostasis]]"

feedback2="'''Incorrect.''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: calcium homeostasis]]."

feedback2="'''Incorrect.''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: Calcium homeostasis]]"

feedback1="'''Incorrect.''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: calcium homeostasis]]."

feedback1="'''Incorrect.''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: Calcium homeostasis]]"

feedback4="'''Incorrect.''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: calcium homeostasis]]."

feedback4="'''Incorrect.''' PTH increases the synthesis of calcitriol by enhancing the 1alpha- hydroxylation reaction in the kidney. [[Calcium Homeostasis - Anatomy & Physiology|WikiVet Article: Calcium homeostasis]]"

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</WikiQuiz>  

</WikiQuiz>  

choice5="Zona reticularis of cortex"

choice5="Zona reticularis of cortex"

correctchoice="3"

correctchoice="3"

feedback3="'''Correct!''' The adrenal medulla produces catecholamines including epinephrine and norepinephrine. [[Adrenal Glands - Anatomy & Physiology|WikiVet Article: adrenal glands]]."

feedback3="'''Correct!''' The adrenal medulla produces catecholamines including epinephrine and norepinephrine. [[Adrenal Glands - Anatomy & Physiology|WikiVet Article: Adrenal glands]]"

feedback4="'''Incorrect.''' The capsule has no endocrine function. The adrenal medulla produces catecholamines including epinephrine and norepinephrine. [[Adrenal Glands - Anatomy & Physiology|WikiVet Article: adrenal glands]]."

feedback4="'''Incorrect.''' The capsule has no endocrine function. The adrenal medulla produces catecholamines including epinephrine and norepinephrine. [[Adrenal Glands - Anatomy & Physiology|WikiVet Article: Adrenal glands]]"

feedback1="'''Incorrect.''' The zona glomerulosa in the adrenal cortex produces mineralocorticoids e.g. aldosterone. The adrenal medulla produces catecholamines including epinephrine and norepinephrine. [[Adrenal Glands - Anatomy & Physiology|WikiVet Article: adrenal glands]]."

feedback1="'''Incorrect.''' The zona glomerulosa in the adrenal cortex produces mineralocorticoids e.g. aldosterone. The adrenal medulla produces catecholamines including epinephrine and norepinephrine. [[Adrenal Glands - Anatomy & Physiology|WikiVet Article: Adrenal glands]]"

feedback2="'''Incorrect.''' The zona fasciculata in the adrenal cortex produces glucocorticoids e.g. cortisol. The adrenal medulla produces catecholamines including epinephrine and norepinephrine. [[Adrenal Glands - Anatomy & Physiology|WikiVet Article: adrenal glands]]."

feedback2="'''Incorrect.''' The zona fasciculata in the adrenal cortex produces glucocorticoids e.g. cortisol. The adrenal medulla produces catecholamines including epinephrine and norepinephrine. [[Adrenal Glands - Anatomy & Physiology|WikiVet Article: Adrenal glands]]"

feedback5="'''Incorrect.''' The zona reticularis in the adrenal cortex produces adrenal androgens. The adrenal medulla produces catecholamines including epinephrine and norepinephrine. [[Adrenal Glands - Anatomy & Physiology|WikiVet Article: adrenal glands]]."

feedback5="'''Incorrect.''' The zona reticularis in the adrenal cortex produces adrenal androgens. The adrenal medulla produces catecholamines including epinephrine and norepinephrine. [[Adrenal Glands - Anatomy & Physiology|WikiVet Article: Adrenal glands]]"

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</WikiQuiz>

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