Changes

choice2="Glucocorticoids, insulin and thyroxine (T4)"

choice2="Glucocorticoids, insulin and thyroxine (T4)"

correctchoice="5"

correctchoice="5"

feedback5="'''Correct!''' Growth hormone is the main regulator of IGF-I production in the liver. Insulin and oestradiol are stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]."

feedback5="'''Correct!''' Growth hormone is the main regulator of IGF-I production in the liver. Insulin and oestradiol are stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]"

feedback4="'''Incorrect.''' Thyroxine (T4) does not directly affect IGF-I production. However growth hormone is the main regulator of IGF-I production in the liver and insulin is stimulatory in other tissues. The missing hormone is oestradiol which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]."

feedback4="'''Incorrect.''' Thyroxine (T4) does not directly affect IGF-I production. However growth hormone is the main regulator of IGF-I production in the liver and insulin is stimulatory in other tissues. The missing hormone is oestradiol which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]"

feedback3="'''Incorrect.''' Growth hormone is the main regulator of IGF-I production in the liver and insulin is stimulatory in other tissues. Glucocorticoids are inhibitory in other tissues. The missing hormone is oestradiol which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]."

feedback3="'''Incorrect.''' Growth hormone is the main regulator of IGF-I production in the liver and insulin is stimulatory in other tissues. Glucocorticoids are inhibitory in other tissues. The missing hormone is oestradiol which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]"

feedback1="'''Incorrect.''' Growth hormone is the main regulator of IGF-I production in the liver and oestradiol is stimulatory in other tissues. Glucocorticoids are inhibitory in other tissues. The missing hormone is insulin which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]."

feedback1="'''Incorrect.''' Growth hormone is the main regulator of IGF-I production in the liver and oestradiol is stimulatory in other tissues. Glucocorticoids are inhibitory in other tissues. The missing hormone is insulin which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]"

feedback2="'''Incorrect.''' Insulin is stimulatory in many tissues and glucocorticoids are inhibitory in several tissues. Thyroxine (T4) does not directly affect IGF-I production.The missing hormones are growth hormone which is the main regulator of IGF-I production in the liver and oestradiol which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]."

feedback2="'''Incorrect.''' Insulin is stimulatory in many tissues and glucocorticoids are inhibitory in several tissues. Thyroxine (T4) does not directly affect IGF-I production.The missing hormones are growth hormone which is the main regulator of IGF-I production in the liver and oestradiol which is also stimulatory in other tissues. [[IGF-1 - Anatomy & Physiology|WikiVet Article: Insulin-like growth factor]]"

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</WikiQuiz>  

</WikiQuiz>  

choice2="Kidney"

choice2="Kidney"

correctchoice="4"

correctchoice="4"

feedback4="'''Correct!''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: active vitamin D synthesis]]."

feedback4="'''Correct!''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]."

feedback5="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: active vitamin D synthesis]]."

feedback5="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]."

feedback3="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: active vitamin D synthesis]]."

feedback3="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]."

feedback1="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: active vitamin D synthesis]]."

feedback1="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]."

feedback2="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: active vitamin D synthesis]]."

feedback2="'''Incorrect.''' Vitamin D3 is absorbed from the intestine or formed in the skin by the action of UV light on 7-dehydrocholesterol. It is then converted to 25-OH D3 in the liver by 25-hydroxylase. Next it is converted to its active form, calcitriol in the kidney. Calcitriol stimulates resorption of calcium and phosphate from bone and an increase in the amount of calcium and phosphate absorbed from the intestine. [[Calcium Homeostasis - Anatomy & Physiology#Active Vitamin D Synthesis|WikiVet Article: Active vitamin D synthesis]."

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image= "">

</WikiQuiz>  

</WikiQuiz>