Changes

Intussusception results from abnormal peristalsis. Vigorous contractions force a segment of intestine to teloscope into the lumen of a more relaxed adjacent segment.  A normograde intussusception is most common, but retrograde intussusception has also been reported.   

Intussusception results from abnormal peristalsis. Vigorous contractions force a segment of intestine to teloscope into the lumen of a more relaxed adjacent segment.  A normograde intussusception is most common, but retrograde intussusception has also been reported.   

Intussusceptions can be classified according to their location in the gastrointestinal tract. They occur more commonly in regions where there is a significant change in lumen diameter, such as ileocolic and gastroesphageal junctions. Ileocolic intussusceptions are most common they frequently protrude from the rectum and must be distinguished from a rectal prolapse. In the case of an intussusception, it is possible to pass a probe next to the anus, but not in [[Rectal Prolapse|rectal proplapse]].

Intussusceptions can be classified according to their location in the gastrointestinal tract. They usually occur in regions where there is a significant change in lumen diameter, such as ileocolic and gastroesphageal junctions. Ileocolic intussusceptions are most common, they frequently protrude from the rectum and must be distinguished from a rectal prolapse. In the case of an intussusception, it is possible to pass a probe next to the anus, but not in [[Rectal Prolapse|rectal proplapse]].

Initially, a partial obstruction results.  Overtime, this progresses to a complete obstruction, with obstruction of venous return, arterial occlusion and avulsion of vessels.  The intestinal walls become oedematous, ischaemic and turgid, resulting in devitalisation if not treated.  Adhesion can occur in long standing cases due to fibrin deposition.

Initially, a partial obstruction results.  Overtime, this progresses to a complete obstruction, with obstruction of venous return, arterial occlusion and avulsion of vessels.  The intestinal walls become oedematous, ischaemic and turgid, resulting in devitalisation if not treated.  Adhesion can occur in long standing cases due to fibrin deposition.

[[Category:Intestine_-_Physical_Disturbances]][[Category:To_Do_-_Clinical]]

[[Category:Intestine_-_Physical_Disturbances]][[Category:To_Do_-_Clinical]]

[[Category:To_Do_-_lizzyk]]

[[Category:To_Do_-_lizzyk]]