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Although VEE does not cause as high a mortality as WEE/EEE, the clinical signs are similar. However, a second generalised infection may be caused by the virus causing fever, depression, colic and diarrhoea.
 
Although VEE does not cause as high a mortality as WEE/EEE, the clinical signs are similar. However, a second generalised infection may be caused by the virus causing fever, depression, colic and diarrhoea.
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Worse in unvaccinated animals.  Acute signs of EEE and WEE are nonspecific, last up to 5 days and include:
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Worse in unvaccinated animals.   
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=====EEE and WEE=====
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Acute signs are nonspecific and last up to 5 days:
 
*mild to severe pyrexia
 
*mild to severe pyrexia
 
*anorexia
 
*anorexia
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*paralysis of pharynx, larynx and tongue
 
*paralysis of pharynx, larynx and tongue
 
*recumbency for 1-7 days followed by death
 
*recumbency for 1-7 days followed by death
VEE may have similar or different clinical presentations to WEE and EEE, which may relate to a persistently hightitre viraemia with VEE and differences in strain pathogenicity:
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*pyrexia peaks early and remains high throuhgout the disease course
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=====VEE=====
*mild fever and leukopenia associated experimentally with endemic strains
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Clinical signs may resemble those of WEE and EEE or may vary:
*severe pyrexia and leukopenia associated with epidemic strains
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*pyrexia peaks early and persists throughout the disease course
*diarrhoea, severe depression recumbency and death may precede neurological signs
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*mild pyrexia and leukopenia with endemic strains  
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*severe pyrexia and leukopenia with epidemic strains
 
*neurological signs around 4 days post-infection
 
*neurological signs around 4 days post-infection
*other associated signs: abortion, oral ulceration, pulmonary haemorrhage, epistaxis
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*diarrhoea, severe depression, recumbency and death may precede neurological signs
 
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*other signs: abortion, oral ulceration, pulmonary haemorrhage, epistaxis
 
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*Paralysis of the lips
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*Drooping eyelids
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*Incoordination
      
====Diagnosis====
 
====Diagnosis====
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