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The pathogenesis of ''C.jejuni'' involves the colonisation, attachment and invasion of colonic enterocytes and toxin production.  

The pathogenesis of ''C.jejuni'' involves the colonisation, attachment and invasion of colonic enterocytes and toxin production.  

It causes colitis, characterised by; Necrosis of absorptive epithelial cells; Erosion of the mucosa; Crypt abscesses; The infiltration of inflammatory cells into the mucosa and the presence of primarily [[Neutrophils|neutrophils]].

It causes colitis, characterised by; Necrosis of absorptive epithelial cells; Erosion of the mucosa; Crypt abscesses; The infiltration of inflammatory cells into the mucosa and the presence of primarily [[Neutrophils|neutrophils]].

The bacteria's functional flagella are important as virulence factors. Non-flagellate [[Campylobacter species- Overview|campylobacter]] do not colonise in vivo, and are less invasive.

The bacteria's functional flagella are important as virulence factors. Non-flagellate [[Campylobacter species - Overview|campylobacter]] do not colonise in vivo, and are less invasive.

It causes enteritis and diarrhoea in susceptible dogs and causes abortion in ewes.

It causes enteritis and diarrhoea in susceptible dogs and causes abortion in ewes.

It usually causes asymptomatic infections in chickens and turkeys, but occasional outbreaks of avian [[ Hepatitis, Acute|hepatitis]] occur.  

It usually causes asymptomatic infections in chickens and turkeys, but occasional outbreaks of avian [[ Hepatitis, Acute|hepatitis]] occur.