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Equine Protozoal Myeloencephalitis (view source)
Revision as of 00:34, 17 July 2010
, 00:34, 17 July 2010no edit summary
====Differential Diagnoses====
====Differential Diagnoses====
The disease may mimic almost any neurological disease because the parasite can localise in any region of the central nervous system (CNS).(EPM 8)
Includes virtually all diseases of the CNS (Furr)
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|PCR (Furr)
|PCR (Furr)
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|Rabies
|Rapid progression (Sommardahl), behavioural alterations, depression, seizure, coma (Long)
|Post-mortem dx (Sommardahl)
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|Polyneuritis equi
|Cranial nerve deficits peripheral with no change in attitude (6 p623 in Saville)
|Western blot analysis of CSF(20 p623 in Saville)
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|Equine degenerative myeloencephalopathy
|Symmetrical signs (Nout, p606)
|May get increased CSF CK (3 in Nout p608) and reduced serum Vitamin E concentrations but unreliable ante mortem dx
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|Verminous encephalomyelitis
|Acute onset
|CSF analysis(Jose-Cunilleras)
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|Bacterial meningoencephalitis
|Stiff neck (Pasq)
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|CNS abscessation (Furr)
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|Spinal trauma(Pasq)
|Hx (usually acute onest nuero signs), usually spolitary lesion loclaised by neuro exa (71 p589)
|Rads, myelography, CT, MRI, nuclear scintigraphy, CSF analysis, nerve conduction velocities, EMG, transcranial magnetic stimulation (p590)
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|Occipito-atlanto-axial malformation
|Deficits develop before 6mths (7,12, Seino)
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|Spinal tumors
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|CT, MRI, definitive dx requires cytology, biopsy, histopathology, CSF analysis (Sellon)
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|Sorghum cystitis/ataxia (Pasq)
|Posterior ataxia or paresis, cystitis, hx of grazing Sorghum species (Talcott, ch22
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Cauda equina neuritis
Cauda equina neuritis
====Prognosis====
====Prognosis====