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Affected animals may show few clinical signs as the condition first develops but, in more advanced cases, the following signs may be documented:
 
Affected animals may show few clinical signs as the condition first develops but, in more advanced cases, the following signs may be documented:
 
*'''Anorexia''' and severe '''weight loss'''
 
*'''Anorexia''' and severe '''weight loss'''
*[[Control of Feeding - Anatomy & Physiology#The Vomit Reflex|'''Vomiting''']] which may contain blood if blood vessels are eroded ('''haematemesis''').  Vomiting occurs due to gastric irritation and possible gastric outflow obstruction.   
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*[[Vomiting|'''Vomiting''']] which may contain blood if blood vessels are eroded ('''haematemesis''').  Vomiting occurs due to gastric irritation and possible gastric outflow obstruction.   
 
*'''[[Diarrhoea]]''' which may show evidence of haemorrhage in the upper gastro-intestinal tract ('''melaena''').  Diarrhoea occurs due to the production of excessive volumes of gastric secretions and because gastrin reduces the ability of the intestine to absorb fluid and electrolytes.  Malabsorption may occur due to small intestinal villous atrophy.
 
*'''[[Diarrhoea]]''' which may show evidence of haemorrhage in the upper gastro-intestinal tract ('''melaena''').  Diarrhoea occurs due to the production of excessive volumes of gastric secretions and because gastrin reduces the ability of the intestine to absorb fluid and electrolytes.  Malabsorption may occur due to small intestinal villous atrophy.
 
*'''Collapse''' and '''shock''' if an ulcer perforates to cause peritonitis.
 
*'''Collapse''' and '''shock''' if an ulcer perforates to cause peritonitis.
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