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==Description==
==Description==
Development of the '''typical diamond shaped lesion''' on the skin is '''pathognomic''' for this disease. Erysipelas should be considered in any cases where '''fever and lameness occur together''', or in pregnant sows struggling to farrow in combination with a fever.
Development of the '''typical diamond shaped lesion''' on the skin is '''pathognomic''' for this disease. Erysipelas should be considered in any cases where '''fever and lameness occur together''', or in pregnant sows struggling to farrow in combination with a fever.
==Laboratory Tests==
===Clinical Signs===
==Clinical Signs==
Clinical outcome depends on the susceptibility of the pig and the virulence of the strain of Erysipelas. Pigs are susceptible after maternal antibody has waned (after 3 months) and before protective immunity is acquired (3 years).
Clinical outcome depends on the susceptibility of the pig and the virulence of the strain of Erysipelas. Pigs are susceptible after maternal antibody has waned (after 3 months) and before protective immunity is acquired (3 years).
Changes in diet, extremes of temperature and fatigue are thought to predispose to infection.
Changes in diet, extremes of temperature and fatigue are thought to predispose to infection.
3 forms of disease occur: Hyperacute, Acute and Chronic.
3 forms of disease occur: Hyperacute, Acute and Chronic.
The hyperacute form is more common in younger pigs, who may present dull, collapsed or pyrexic with a scarlet tinge to the skin. This form can also present with cases of sudden death.
The '''hyperacute form''' is more common in younger pigs, who may present dull, collapsed or pyrexic with a scarlet tinge to the skin. This form can also present with cases of sudden death.
In the acute form, anorexia and pyrexia are the two most common clinical signs in younger pigs. Affected animals still die but normally after a day during which time they become dyspnoic. Older pigs tend to be pyrexic, anorexic and polydypsic.
In the '''acute form''', anorexia and pyrexia are the two most common clinical signs in younger pigs. Affected animals still die but normally after a day during which time they become dyspnoic. Older pigs tend to be pyrexic, anorexic and polydypsic.
All pigs show, pink/purple raised areas or extensive diamond-shaped plaques over the skin within 24-48 hours of developing clinical signs. If pregnant sows are infected at this time they may abort.
All pigs show, pink/purple raised areas or extensive diamond-shaped plaques over the skin within 24-48 hours of developing clinical signs. If pregnant sows are infected at this time they may abort.
Following the chronic form of the disease, affected animals can completely recover, lesions often resolve within a week however they can become necrotic and slough.
Following the '''chronic form''' of the disease, affected animals can completely recover, lesions often resolve within a week however they can become necrotic and slough.
The bacteria localises in joints causing destruction of the synovial membrane lining, hyperaemia, villus formation and lymphocyte and plasma cell infiltration resulting in chronic serofibrinous polyarthritis.
The bacteria localises in joints causing destruction of the synovial membrane lining, hyperaemia, villus formation and lymphocyte and plasma cell infiltration resulting in chronic serofibrinous polyarthritis.
Initially joints are hot and swollen leading to stiffness, lameness, non-weight bearing on the affected limbs and eventual ankylosis. Discospondylitis can also occur if animals are chronically affected.
Initially joints are hot and swollen leading to stiffness, lameness, non-weight bearing on the affected limbs and eventual ankylosis. Discospondylitis can also occur if animals are chronically affected.
Additionally valvular lesions may develop in the heart causing chronic valvular [[Endocarditis| endocarditis]], vegetative thrombosis of mitral valves and asymptomatic or congestive heart failure sometimes resulting in sudden death following stress.
Additionally valvular lesions may develop in the heart causing chronic valvular [[Endocarditis| endocarditis]], vegetative thrombosis of mitral valves and asymptomatic or congestive heart failure sometimes resulting in sudden death following stress.
===Laboratory Tests===
Slender Gram-positive rods can be seen on microscopy of acute lesions and filamentous forms in chronic lesions and on smears. High levels of antibody may be found in joint fluid and so may aid diagnosis.
[[ELISA testing|ELISA]] and Polymerase Chain Reaction tests are also available which can be used directly on tissue or following culture.
===Pathology===
Post mortem diagnosis may be needed for definitive diagnosis, in particular the, [[Liver - Anatomy & Physiology| liver]], [[Spleen - Anatomy & Physiology|spleen]], heart valves, [[The Endocrine Function of the Kidney - Anatomy & Physiology| kidney]], a long bone and synovial tissues should be submitted for bacterial examination.
Post mortem diagnosis may be needed for definitive diagnosis, in particular the, [[Liver - Anatomy & Physiology| liver]], [[Spleen - Anatomy & Physiology|spleen]], heart valves, [[The Endocrine Function of the Kidney - Anatomy & Physiology| kidney]], a long bone and synovial tissues should be submitted for bacterial examination.
==Prognosis==
==Prognosis==
Varies from sudden death to recovery depending on the virulence of the strain and age and immune status of the affected pig.
Varies from sudden death to recovery depending on the virulence of the strain and age and immune status of the affected pig.
==References==
==References==
[[Category:Pig]][[Category:To_Do_-_Caz]]
[[Category:Pig]][[Category:To_Do_-_Caz]]
[[Category:To_Do_-_Review]]
[[Category:Expert_Review]]