Changes

==Diagnosis==

==Diagnosis==

The classical lesions of photosensitisation are usually sufficient for diagnosis of primary disease. There is often a history of exposure or ingestion of plants containing photodynamic agents such as St. John's Wort or clover. Additionally, phenothiazines, sulphonamides, tetracyclines and mycotoxins such as blue-green algae have been implicated in photosensitisation.

The classical lesions of photosensitisation are usually sufficient for diagnosis of primary disease. There is often a history of exposure or ingestion of plants containing photodynamic agents such as St. John's Wort or clover. Additionally, phenothiazines, sulphonamides, tetracyclines and mycotoxins such as blue-green algae have been implicated in photosensitisation.

==Treatment==

==Treatment==

Affected animals should only be allowed access to grazing at night. If the animal is affected by secondary photosensitisation, a low-protein, high-energy diet is recommended such as beet pulp.

Affected animals should only be allowed access to grazing at night. If the animal is affected by secondary photosensitisation, a low-protein, high-energy diet is recommended such as beet pulp. Anti-inflammatory drugs such as flunixin meglumine and topical steroid creams may be useful to decrease the severity of cutaneous lesions in the acute stage of the disease. If skin necrosis is severe, surgical debridement may be required.  

==Prognosis==

==Prognosis==