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512 bytes added ,  11:00, 25 August 2010
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patients with abnormalities of either primary or secondary haemostasis may show external haemorrhage such as epistxis, haematemesis, melaena and haematuria.. Sever blood loss may cause hypovolaemia and/or anaemia. Small volumes of blood within a sensitive location (for example, the brain, eyes. spinal cord or pericardial sac) can cause dramatic clinical signs. Many patients with haemostatic defects, however, can appear to be stable or clinically normal.
 
patients with abnormalities of either primary or secondary haemostasis may show external haemorrhage such as epistxis, haematemesis, melaena and haematuria.. Sever blood loss may cause hypovolaemia and/or anaemia. Small volumes of blood within a sensitive location (for example, the brain, eyes. spinal cord or pericardial sac) can cause dramatic clinical signs. Many patients with haemostatic defects, however, can appear to be stable or clinically normal.
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disroders of fibrinolysis may result in thrombus formation and loss of blood supply. The fomation of thrombi is promoted by: local endothelial injury (vascular damage), circulatory stasis and changed in anticoagulants or procoagulants, e.g. decreased ATIII. The most common mechanisms for AT deficiency are glomerular disease (AT is similar in size to albumin and is thus lost via the urine in glomerular diseasesP and accelarated consumption which occurs with disseminated intravascular coagulation or sepsis.
    
==Tests Evaluating Primary Haemostasis==
 
==Tests Evaluating Primary Haemostasis==
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