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==Description==
==Description==
Transudates are a type of [[:Category:Effusions|effusion]] that has a low cellularity and a low protein content. Transudates occur due to imbalances in the [[Blood Pressure & Blood Flow Control - Anatomy & Physiology#Capillary Fluid Shift Mechanism |'''Starling forces''']], of which the two principal examples are hydrostatic pressure and plasma oncotic pressure (or colloid osmotic pressure). The following causes of transudate effusions are therefore recognised:
Transudates are a type of [[:Category:Effusions|effusion]] that has a low cellularity and a low protein content. Transudates occur due to imbalances in the [[Blood Pressure Physiology#Capillary Fluid Shift Mechanism |'''Starling forces''']], of which the two principal examples are hydrostatic pressure and plasma oncotic pressure (or colloid osmotic pressure). The following causes of transudate effusions are therefore recognised:
*'''Reduced plasma oncotic pressure''' may occur if plasma proteins are lost. Albumin contributes 75% of the total oncotic pressure so [[Hypoalbuminaemia|'''hypoalbuminaemia''']] is the major cause of transudate effusions.
*'''Reduced plasma oncotic pressure''' may occur if plasma proteins are lost. Albumin contributes 75% of the total oncotic pressure so [[Hypoalbuminaemia|'''hypoalbuminaemia''']] is the major cause of transudate effusions.
*'''Increased hydrostatic pressure''' may occur with inappropriate activation of the renin-angiotensin-aldosterone system (RAAS), as in congestive heart failure and portal hypertension. Both diseases commonly result in high-volume ascites, although this is more often composed of a [[Modified Transudate|modified transudate]].
*'''Increased hydrostatic pressure''' may occur with inappropriate activation of the renin-angiotensin-aldosterone system (RAAS), as in congestive heart failure and portal hypertension. Both diseases commonly result in high-volume ascites, although this is more often composed of a [[Modified Transudate|modified transudate]].