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| − | <FlashCard questions="14"> | + | <FlashCard questions="6"> |
| − | |q1=Name the virus that causes avian encephalomyeltits (AE)? | + | |q1=Name some of the syndromes associated with avian adenovirus infections? |
| − | |a1=Avian encephalomyelitis-like virus 1 (AEV). | + | |a1= |
| − | |l1=Avian Encephalomyelitis#Introduction | + | *Quail bronchitis (QB) |
| − | |q2= Is avian encephalomyelitis virus (AEV) an RNA or DNA virus? | + | *Egg drop syndrome (EDS) |
| − | |a2= AEV is a single stranded RNA (ssRNA) virus | + | *Haemorhagic Enteritis (HE) |
| − | |l2=Avian Encephalomyelitis#Introduction | + | *Marble Spleen Disease in pheasants (MSD) |
| − | |q3= What genus does AEV belong to? | + | *Inclusion Body Hepatitis (IBH) |
| − | |a3= Hepatovirus | + | |l1=Avian Adenoviruses#Diseases |
| − | |l3=Avian Encephalomyelitis#Introduction | + | |q2= At what age do adenovirus infections usually present and why? |
| − | |q4= What family does the virus belong to? | + | |a2= Around 4-6 weeks in broilers or 5-9 weeks in layers, because this is when maternal immunity wanes. |
| − | |a4=Picornaviridae | + | |l2= Avian Adenoviruses#Diseases |
| − | |l4=Avian Encephalomyelitis#Introduction | + | |q3= What are the common clinical signs of EDS? |
| − | |q5= What are the main economic concerns for breeders and layers about AE? | + | |a3= |
| | + | *Egg drop |
| | + | *Pale/hypopigmented egg shells |
| | + | *Rough, soft, misshapen eggs |
| | + | *Decreased hatchability |
| | + | *Small reproductive tracts in females |
| | + | |l3= Avian Adenoviruses#Clinical Signs |
| | + | |q4= How can EDS be diagnosed that is different to the other adenoviruses? |
| | + | |a4= By haemagglutination inhibition. |
| | + | |l4= Avian Adenoviruses#Diagnosis |
| | + | |q5=How can virus be identified once isolated or innoculated? |
| | |a5= | | |a5= |
| − | *Temporary decrease in egg production in laying hens | + | *Electron miscroscopy |
| − | *Decrease in egg hatchability | + | *Staining with fluorescent labelled antisera |
| − | *Neurological disease and morbidity /mortality of chicks under three weeks of age
| + | |l5= Avian Adenoviruses#Diagnosis |
| − | *Surviving chicks are considered unlikely to be profitable
| + | |q6= Against which two adenoviruses are vaccines available? |
| − | |l5=Avian Encephalomyelitis#Introduction | |
| − | |q6= How is AE spread and what is its epidemiology? | |
| | |a6= | | |a6= |
| − | *Horizontally via faecal-oral route (main route of infection) | + | *EDS |
| − | *Vertical transmission from infected layer to their chicks | + | *HE |
| − | *The virus replicates in the epithial cells of the alimentary tract and is circulated in the bloodstream to other organs and the central nervous system (CNS). The virus is shed within 3 days of oral ingestion and in young chicks can continue to be shed for over two weeks. Shedding ceases once specific antibodies are produced.
| + | |l6= Avian Adenoviruses#Treatment and Control |
| − | |l6= Avian Encephalomyelitis#Introduction | |
| − | |q7= What species of birds does AE affect?
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| − | |a7=
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| − | *Chickens (main domestic host)
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| − | *Species of partridge
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| − | *Turkey
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| − | *Quail
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| − | *Guineafowl
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| − | *Pheasants
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| − | |l7= Avian Encephalomyelitis#Signalment
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| − | |q8=What are the clinical signs of AE?
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| − | |a8= AE causes the most serious disease in chicks under 3 weeks of age. The clinical signs for this age group are:
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| − | *neurological signs ranging from ataxia, rapid tremor of the head and neck, drooping of the wings, weakness, paralysis, exercise intolerance to blindness and changes in vocalisation.
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| − | *Weight loss, lameness, and unthriftiness
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| − | *Affected chicks sit on their hocks, and cannot move well and many fall over onto their sides
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| − | | |
| − | Laying hens rarely show clinical signs but the virus causes:
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| − | *Temporary reduction (2 weeks) in egg production
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| − | *Decrease in droppings
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| − | *Decrease in hatching rates
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| − | |l8= Avian Encephalomyelitis#Clinical Signs
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| − | |q9= How can AE be diagnosed?
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| − | |a9=
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| − | The following methods have been used to help diagnose Avian Encephalomyelitis (AE):
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| − | *Virus Neutralization test
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| − | *Agar Gel test
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| − | *Embryo Susceptibility test
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| − | *Elisa test
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| − | All of the above tests indicate exposure to AE but not neccessarily a current infection.
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| − | Diagnosis can be confirmed by either histopathology or by virus isolation.
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| − | | |
| − | |l9= Avian Encephalomyelitis#Diagnosis
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| − | |q10=What is the distribution of AE?
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| − | |a10=Worldwide, the disease has been documented in Africa, Asia, Australia, Europe, and North and South America.
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| − | |l10=Avian Encephalomyelitis#Distribution
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| − | |q11=What is the treatment for AE?
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| − | |a11=There is no treatment for chicks infected with avian encephalomyelitis (AE).
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| − | |l11=Avian Encephalomyelitis#Treatment
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| − | |q12=Can chicken and surviving chicks become immune to AEV?
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| − | |a12=Yes, surviving chicks, infected hens and birds that have been vaccinated will be immune to AE for life.
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| − | |l12=Avian Encephalomyelitis#Treatment
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| − | |q13=How many serotypes are there for AEV?
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| − | |a13=There is only one serotype for the AEV.
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| − | |l13=Avian Encephalomyelitis#Control
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| − | |q14=How can AE be controlled?
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| − | |a14=Vaccination of pullets several weeks before they come into lay. There are live and attenuated vaccinations available which can be administered in drinking water or as an eye drop, the latter being more effective.
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| − | |l14=Avian Encephalomyelitis#Control
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| | </FlashCard> | | </FlashCard> |
| − | | + | [[Category: Avian Flashcards]] |
| − | | + | [[Category:To Do - Jaimie Meagor]] |
| − | [[Category:Avian Flashcards]] | |
| − | [[Category:CABI Flashcards]] | |
| − | [[Category:Neurological Diseases Flashcards]]
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