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*Non-motile
 
*Non-motile
 
*Catalase negative, oxidase negative, coagulase positive
 
*Catalase negative, oxidase negative, coagulase positive
*Isolates from acute infections produce smooth colonies
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*Isolates from chronic infections produce rough colonies
   
*Grow on nutrient agar; growth enhanced by addition of blood or serum
 
*Grow on nutrient agar; growth enhanced by addition of blood or serum
 
*Small colonies with incomplete haemolysis in 48 hours
 
*Small colonies with incomplete haemolysis in 48 hours
*Hydrogen sulphide sormed along stab line in TSI agar
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*Hydrogen sulphide formed as black central line in TSI agar
 
*Growth over wide rangs of temperatures and pH
 
*Growth over wide rangs of temperatures and pH
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===Identification and diagnosis===
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*Isolates from acute infections produce smooth colonies; 1.5mm diamteter, convex and circular with even edges
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*Isolates from chronic infections produce rough colonies; larger, flat and opaque colonieswith irregular edges
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*Biochemical tests
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*Serotypying according to a heat-stable peptidoglycan extracted from the cell wall
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*Virulence testing in lab animals
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*PCR for detection of virulent isolates
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===Pathogenesis and pathogenicity===
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*Route of infection: ingestion from pig faeces
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*Entry via tonsils, skin or mucous membranes
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*Capsule prevents phagocytosis
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*Adhere to endothelial cells
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*Produce neurominidase which enhances cell penetration
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*Septicaemia with vascular damage
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*Swelling of endothelial cells, monocyte adherence to vascular walls and hyaline microthrombus formation
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*Bacteria localise in synovia of joints and on heart valves during bacteraemia and cause chronic lesions
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*Host immune response to persistent bacterial antigens causes long-term damage to the joints
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===Clinical infections===
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