Difference between revisions of "Placenta Endocrine Function - Anatomy & Physiology"

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<big><center>[[Endocrine System - Anatomy & Physiology|'''BACK TO ENDOCRINE SYSTEM ANATOMY & PHYSIOLOGY''']]</center></big>
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The Placenta is a hugely important endocrine organ, producing many [[Hormones - Anatomy & Physiology|hormones]] which affect the status of pregnancy and the maternal physiology.
 
  
  
 
{| border="1" cellpadding="5" cellspacing="0"
 
{| border="1" cellpadding="5" cellspacing="0"
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|+ ''Hormones of the Placenta''
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|+ ''Hormones of the Testes''
 
!Hormone
 
!Hormone
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!Production and Regulation
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!Regulation
 
!Action
 
!Action
 
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|Prostaglandin F2a
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|<font color=Purple>Prostaglanding F2a</font>
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|Produced towards the end of pregnancy in increasing amounts to allow for parturition.
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|Leydig Cells
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|Causes luteolysis and promotes uterine contractions.
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|GnRH from the [[Endocrine System - Hypothalamus - Anatomy & Physiology|Hypothalamus]] causes LH secretion from the [[Endocrine System - Pituitary Gland - Anatomy & Physiology|Pituitary Gland]] which stimulates the Leydig Cells.
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|This hormone controls and maintains the growth and functions of the reproductive organs. It enhances libido and is essential for spermatogenesis.
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|Progesterone
 
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|The Placenta takes over Progesterone production from the Corpus Luteum to maintain the pregnancy.
 
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|Maintains the pregnancy by thickening the cervix, depressing the maternal immune response and preventing ovulation. It also promotes mammary growth.
 
 
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|Oestrogen
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|<font color=Purple>Progesterone</font>
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|Oestrogen levels rise throughout pregnancy as the placenta grows.
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|Sertoli Cells
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|Stimulate the growth of the myometrium, promote mammary development and suppress gonadotrophin release from the [[Pituitary Gland - Anatomy & Physiology|pituitary gland]].
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|GnRH from the [[Endocrine System - Hypothalamus - Anatomy & Physiology|Hypothalamus]] causes FSH secretion from the [[Endocrine System - Pituitary Gland - Anatomy & Physiology|Pituitary Gland]] which stimulates the Sertoli Cells.
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|Prevents secretion of further FSH from the [[Endocrine System - Pituitary Gland - Anatomy & Physiology|Pituitary Gland]].
 
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|Placental Lactogen
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|<font color=Purple>Oestrogen<font>
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|This is related to [[Lactation - Endocrine Control - Anatomy & Physiology|Prolactin]] and [[Pituitary Growth Hormone - Anatomy & Physiology|Growth hormone]], and is produced by the placenta and uterus.
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|Sertoli Cells
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|Promotes growth and differentiation of mammary tissue. Identified in primates, ruminants and rodents.
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|GnRH from the [[Endocrine System - Hypothalamus - Anatomy & Physiology|Hypothalamus]] causes FSH secretion from the [[Endocrine System - Pituitary Gland - Anatomy & Physiology|Pituitary Gland]] which stimulates the Sertoli Cells.
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|Converted from testosterone, this hormone's function is complex. It may prevent apoptosis of male germ cells.
 
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|Equine Chorionic Gonadotropin (eCG)
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|<font color=Purple>Equine Chorionic Gonadotropin (eCG)<font>
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|This is produced by foetal trophoblast cells, and is actually the same molecule as equine LH.
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|Sertoli Cells
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|Prevents luteal regression and is thus the signal for [[Maternal Recognition of Pregnancy - Anatomy & Physiology|Maternal Recognition of Pregnancy]] in equids.
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|GnRH from the [[Endocrine System - Hypothalamus - Anatomy & Physiology|Hypothalamus]] causes FSH secretion from the [[Endocrine System - Pituitary Gland - Anatomy & Physiology|Pituitary Gland]] which stimulates the Sertoli Cells.
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|Converted from testosterone, this hormone's function is complex. It may prevent apoptosis of male germ cells.
 
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|Human Chorionic Gonadotropin (hCG)
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|<font color=Purple>hCG<font>
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|This is produced by foetal trophoblast cells.
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|Sertoli Cells
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|Prevents luteal regression and is thus the signal for [[Maternal Recognition of Pregnancy - Anatomy & Physiology|Maternal Recognition of Pregnancy]] in primates.
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|GnRH from the [[Endocrine System - Hypothalamus - Anatomy & Physiology|Hypothalamus]] causes FSH secretion from the [[Endocrine System - Pituitary Gland - Anatomy & Physiology|Pituitary Gland]] which stimulates the Sertoli Cells.
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|Converted from testosterone, this hormone's function is complex. It may prevent apoptosis of male germ cells.
 
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==Webinars==
 
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<rss max="10" highlight="none">https://www.thewebinarvet.com/urogenital-and-reproduction/webinars/feed</rss>
 
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[[Category:Endocrine System - Anatomy & Physiology]][[Category:Pregnancy and Parturition]]
 

Revision as of 11:19, 3 July 2008

BACK TO ENDOCRINE SYSTEM ANATOMY & PHYSIOLOGY



Hormones of the Testes
Hormone Regulation Action
Prostaglanding F2a Leydig Cells GnRH from the Hypothalamus causes LH secretion from the Pituitary Gland which stimulates the Leydig Cells. This hormone controls and maintains the growth and functions of the reproductive organs. It enhances libido and is essential for spermatogenesis.
Progesterone Sertoli Cells GnRH from the Hypothalamus causes FSH secretion from the Pituitary Gland which stimulates the Sertoli Cells. Prevents secretion of further FSH from the Pituitary Gland.
Oestrogen Sertoli Cells GnRH from the Hypothalamus causes FSH secretion from the Pituitary Gland which stimulates the Sertoli Cells. Converted from testosterone, this hormone's function is complex. It may prevent apoptosis of male germ cells.
Equine Chorionic Gonadotropin (eCG) Sertoli Cells GnRH from the Hypothalamus causes FSH secretion from the Pituitary Gland which stimulates the Sertoli Cells. Converted from testosterone, this hormone's function is complex. It may prevent apoptosis of male germ cells.
hCG Sertoli Cells GnRH from the Hypothalamus causes FSH secretion from the Pituitary Gland which stimulates the Sertoli Cells. Converted from testosterone, this hormone's function is complex. It may prevent apoptosis of male germ cells.
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