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Frohlich's Adiposogenital Syndrome (view source)
Revision as of 17:11, 5 September 2008
, 17:11, 5 September 2008New page: {{toplink |backcolour = FAFAD2 |linkpage =Endocrine System - Pathology |linktext =Endocrine System |maplink = Endocrine System (Content Map) - Pathology |pagetype =Pathology |sublink1=Pitu...
{{toplink
|backcolour = FAFAD2
|linkpage =Endocrine System - Pathology
|linktext =Endocrine System
|maplink = Endocrine System (Content Map) - Pathology
|pagetype =Pathology
|sublink1=Pituitary Gland - Pathology
|subtext1=PITUITARY GLAND
}}
<br>
Clinical signs due to a hypothalamic dysfunction.
Decresaed synthesis and release of GnRH means that the pituitary is not stimulated to release FSH or LH at normal levels. Clincally seen as:
*Small, atrophic genitals.
*Feminisation in the male due to lower testosterone release.
Hypothalamus damage also allows incresaed appetite and the animal will be:
*Obese.
*Excess deposition of sub-cut fat.
Compression of local structures also leads to:
*Mental retartdation.
*Diabetes insipidus.
*Visual impairment.
|backcolour = FAFAD2
|linkpage =Endocrine System - Pathology
|linktext =Endocrine System
|maplink = Endocrine System (Content Map) - Pathology
|pagetype =Pathology
|sublink1=Pituitary Gland - Pathology
|subtext1=PITUITARY GLAND
}}
<br>
Clinical signs due to a hypothalamic dysfunction.
Decresaed synthesis and release of GnRH means that the pituitary is not stimulated to release FSH or LH at normal levels. Clincally seen as:
*Small, atrophic genitals.
*Feminisation in the male due to lower testosterone release.
Hypothalamus damage also allows incresaed appetite and the animal will be:
*Obese.
*Excess deposition of sub-cut fat.
Compression of local structures also leads to:
*Mental retartdation.
*Diabetes insipidus.
*Visual impairment.